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Query: UMLS:C0271276 (Hudson)
 1,066 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

Evolutionary theory predicts that very young mothers would be more likely to kill an infant than older women, given that the younger mother has a much greater ability to "replace" the dead child through subsequent pregnancies and thus to produce offspring for the next generation. Evolutionary theory also predicts that a woman would be more likely to kill a child if the child was obviously defective, the pregnancy was the result of incest or rape, or if the mother's means of supporting the child were severely compromised. The authors hypothesized that mentally ill mothers would behave in a way that differed significantly from evolutionary expectations, i.e., that they would be more likely to kill children who were older than those killed by mothers in the general population and that the mothers themselves would be likely to be older than mothers in the general population when the murders occurred. To test this hypothesis, the authors compared infanticides (both filicides and neonaticides) committed by mentally ill mothers with those committed by mothers in the general population. They examined two samples: 1) all cases of maternal infanticide from the Mid-Hudson Forensic Psychiatric Hospital from 1978 (when the hospital began admitting female patients) through the year 2000 and 2) a general population sample from a 10-year Canadian study reported by Daly and Wilson in 1998. The authors focused on the following variables: ages of the mothers, ages of the child-victims, whether the pregnancy was the result of rape or incest, whether the child had significant behavioral or physical problems, and whether there were problems supporting the child (e.g., having no partner, poverty, mother's lack of education). The results of the analyses supported the authors' hypothesis about ages of mothers and children. The mentally ill mothers in the Mid-Hudson sample were generally older when they killed their children and the children who were killed were generally older than in the Daly and Wilson general population sample (where the majority of the cases involved neonaticide and the mothers were generally younger than 25 years of age). The three factors, poverty, low education level (or low intellectual capacity), and lack of a spouse were common in both samples. Findings concerning cultural factors, motives, and methods used will be presented in separate publications.
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PMID:Infanticide in female forensic patients: the view from the evolutionary standpoint. 1565 Jun 20

T. Ward and S. M. Hudson (1998) have proposed a self-regulation model of the offence process which is specific to sexual offenders and which attempts to account for the deficiencies in the traditional relapse prevention model as applied to this group of offenders. The self-regulation model is a nine-stage process of offending that addresses both the individual's goals with respect to the offending behavior (approach versus avoidance) and the manner in which the individual attempts to achieve these goals (passive versus active), resulting in four hypothesized pathways that lead to sexual offending. The present study evaluated the validity of this model with a sample of adult male sexual offenders (N=80) treated within the Correctional Service of Canada. Results demonstrated support for the self-regulation model. Specifically, it was found that the four pathways contained in this model were differentially associated with offender types (e.g., incest offender, rapist, extrafamilial child molester, etc.). In addition, static and dynamic risk factors were found to vary among the four pathways in predicted directions and are consistent with the theoretical model. Finally, static and dynamic risk factors differentially predicted pathway membership, again in the expected directions. Implications of findings and the self-regulation model for the assessment and treatment of sexual offenders are discussed.
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PMID:The self-regulation model of sexual offending: the relationship between offence pathways and static and dynamic sexual offence risk. 1687 49