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Query: UMLS:C0220723 (PCA)
 4,687 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

The authors studied changes of LV dP/dt during transcutaneous coronary angioplasty (TCA). The aim of the study was to detect the alterations of LV function during coronary occlusion and to evaluate the immediate effects of PCA on myocardial function. Six patients with incapacitating angina and isolated left anterior descending disease were successfully treated by TCA using Gruntzig's technique. The study protocol included several recording sequences per patient during the phases of balloon inflation at progressively increasing pressures from 2 to 10 hours. Each sequence comprised a recording under basal conditions and every 5 seconds during inflation (20 seconds) and deflation (45 seconds) of the following parameters: heart rate, aortic and LV pressures, positive and negative peaks of LV dP/dt, and the intracoronary pressure gradient at the beginning and the end of each sequence. The first part of the results based on 27 recorded sequences analysed the bad effects of myocardial ischaemia; coronary occlusion induced a significant fall (p less than 0.01) in the positive and negative peak dP/dt values and on elevation (p less than 0.01) in LV end diastolic pressure, without affecting LV systolic pressure or heart rate. These changes have the following characteristics: they are early, occurring within seconds of coronary occlusion; they affect LV contraction and relaxation simultaneously, but the effects are more marked on LV relaxation; the severity is proportional to the duration of occlusion; they are totally reversible; the disturbances of relaxation return to normal more quickly than those of contraction.(ABSTRACT TRUNCATED AT 250 WORDS)
Arch Mal Coeur Vaiss 1985 Mar
PMID:[Changes in left ventricular dP/dt during transluminal coronary angioplasty]. 315 68

Vessels of base of skull were inaccessible to conventional ultrasonic image until 1982, when the use of pulsed Doppler emitting a beam of 2 MHz coupled with a frequency analyzer allowed direct exploration of terminal branches of the internal carotid artery (ICA) and the basilar trunk. It is now possible to measure rate of flow in middle cerebral artery (MCA), anterior (ACA) and posterior (PCA) cerebral arteries, communicating arteries and those of basilar trunk. Practical applications of this new method are numerous in cerebrovascular disease: diagnosis of brain stem lesions, evaluation of effects of extracranial lesions, detection and follow up of arteriovenous malformations, functional value of the circle of Willis prior to carotid surgery. Despite certain limitations due to anatomic factors, angiographic confrontations attest the value and reliability of this new examination.
J Mal Vasc 1988
PMID:[Transcranial Doppler pulse. Methodology, diagnostic value and limitations]. 329 25

The authors report the case of chronic destructive polychondritis (PCA) with extensive and predominant laryngo-tracheal involvement. The respiratory localisation of PCA is apparent in around 50% of cases. In a quarter of patients presenting with this type of localisation, the outcome is fatal. Tracheal disorders as the presenting feature is a rarity, as in the majority of cases it occurs either concomitantly or after the appearance of classic cartilagenous disease in the nose or ear. The diagnosis of the airways disease rests on the interpretation of the flow volume curve and the CT scan of the inspiration and expiration. Taking account of the absence of any correlation between the disturbed respiratory function and the laryngotracheal bronchial lesions, the authors stress the value of an endoscopic assessment with a fibroscope of reduced calibre. The narrowing of the laryngo-tracheal region is due to a thickening of the walls with oedema and inflammatory granulomas, and to collapse of the lumen caused by the destruction of cartilage and by fibrous scarring of the tracheal walls. Either general or local treatment is virtually non-existent and the prognosis of these extensive respiratory forms of the disease is very poor.
Rev Mal Respir 1993
PMID:[Chronic atrophic polychondritis. Clinical polymorphism and severity of respiratory lesions]. 834 70