Gene/Protein Disease Symptom Drug Enzyme Compound
Pivot Concepts:   Target Concepts:
Query: UMLS:C0040822 (tremor)
 18,428 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

In a 50-year-old woman, working as a dental assistant for more than ten years, chronic mercury poisoning developed insidiously, apparently from careless handling of mercury-amalgam. The main signs consisted of mental and neurological changes such as erethism, tremor and mercurial psellism. Peripheral arterial circulatory disorders occurred in the course of the disease, as well as abdominal colic and a polyneuropathy, which provided the first clues to panarteritis nodosa subsequently confirmed histologically.
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PMID:[An unusual case of panarteritis nodosa associated with chronic mercury poisoning (author's transl)]. 1 86

We present the clinical case of a patient with vascular compression of the duodenum or superior mesenteric artery compression syndrome.A female, 42 years old, with history of two months' evolution characterized by postprandial epigastric colic, without irradiation, accompanied by nausea and intractable vomiting, weight loss and gastric shaking. A double contrast gastric duodenum x-ray showed the duodenal frame with exaggerated dilatation and stenosis close to the Treitz angle, through which the contrast media barely flowed. The endoscopy revealed duodenal obstruction, gastric retention and erosive esophagitis. The computerized tomography identified a significant dilatation of the duodenal arc, with stenosis on the aorto-mesenteric junction. We performed an exploratory laparotomy, making a latero-lateral duodenojejunal trans-mesocolic anastomosis. Satisfactory evolution and discharge without complications.
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PMID:[Wilkie's syndrome: vascular duodenal compression]. 1237 20

The devastating and for the most part irremediable consequences for an infant, his or her family, and society in cases of abusive head trauma have spurred research into ways of preventing it. In the last four or five decades, increasing interest in infant crying and its clinical manifestation of colic has led to a reconceptualization of crying in early infancy, such that most of the characteristics of colic can be understood as manifestations of the crying typical of normal infants. This includes an early increase and then decrease in the amount of crying, the unexpected and unpredictable appearance of prolonged crying bouts, and the presence of inconsolable crying that occurs in the early months of life. When these concepts are merged with anecdotal clinical experiences, perpetrator confessions and epidemiological evidence of abusive head trauma, it is clear that these crying characteristics--and caregiver responses--are the predominant, and potentially modifiable, risk factors for abusive head trauma. This unfortunate but understandable relationship between early crying, shaking and abuse has opened windows of opportunity for primary, universal prevention efforts that are appropriate for--and support--all parents and may be able to prevent at least some of these tragic cases.
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PMID:Crying as a trigger for abusive head trauma: a key to prevention. 2550 27