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Query: UMLS:C0040586 (tracheobronchitis)
449 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

Environmental lung injury may take the form of acute tracheobronchitis, asthma, pulmonary edema, chronic bronchitis, emphysema, allergic pneumonitis, fibrosing alveolitis, pleurisy, and neoplastic disease. Environmental factors eliciting these responses include irritant gases and fumes, oxidants, organic allergens, inorganic dust, bacterial enzymes, and high partial pressures of oxygen. The basic pulmonary reactions to these toxic agents--bronchoconstriction, vasoconstriction, increased vascular permeability, inflammation, carcinogenesis--may be mediated, aggravated, or modulated by biologically active substances. These humoral agents include biogenic amines (e.g. histamine): peptides (e.g., bradykinin, vasoactive intestinal peptide, and spasmogenic lung peptide); enzymes (e.g., proteases, superoxide dismutase, and mixed function oxidases); and acidic lipids (e.g., prostaglandins, prostaglandin endoperoxides, and thromboxanes).
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PMID:Environmental injury of the lung: role of humoral mediators. 35 83

The report analyses clinical manifestations, preoperative preparatory procedures, variants and results of urgent and emergency surgical procedures carried out in 47 cases of advanced thyroid cancer. Syndrome of grave lung-heart failure proved a major clinical sign of the disease. It could be compensated by intensive care (in a special ward included). Cervical and cervico-transsternal access was used, and surgery was performed to an extent determined by the pattern and extension of tumor. Thirteen patients died within a few days after operation and 23 at later stages. Among immediate causes of death were lung artery embolism, obstructive tracheobronchitis and tumor progression. Development of emergencies was avoided in many cases by carrying out timely examination and treatment.
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PMID:[Emergency and urgent operations in the treatment of patients with thyroid cancer]. 246 Oct

Management of 150 patients with lung cancer after pneumonectomy has shown that the basic factors causing the onset of postoperative pleural empyema are chronic nonspecific pulmonary diseases, pronounced obstructive syndrome and right heart overload, tumor histology, local tumor size and cancer complications, duration and techniques of surgery, duration of controlled lung ventilation, as well as the onset of postoperative suppurative tracheobronchitis and surgical wound suppuration. The likelihood of empyema development may be assessed only upon analysis of the combination of risk factors with reference to their low informative value, preoperative patients' condition and intraoperative factors which have an equal impact on the onset of this complication. The efficacy of the algorithm elaborated is as follows: if the likelihood of empyema development exceeds 50%, it occurs in 96.3% of cases, if the likelihood of the complication is less than 10%, the postoperative period is uncomplicated in 87.8% of patients.
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PMID:[Determination of likelihood of the development of pleural empyema in patients with cancer after pneumonectomy]. 794 1

Bronchial mucoepidermoid carcinoma is a rare malignancy with varied disease courses.The symptoms are non-specific and mostly related to infection and airway obstruction secondary to the tumor.Bronchial mucoepidermoid carcinoma is difficult to be distinguished from pneumonia,tracheobronchitis,chronicobstructive pulmonary disease,asthma,and other respiratory diseases in its early stage.The age of onset is mostly between 20 and 55 years,and the disease is rarely seen in children.Confirmation of bronchial mucoepidermoid carcinoma depends on bronchoscopy and pathological biopsy,and this disease is often missed and misdiagnoed.
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PMID:[Bronchial Mucoepidermoid Carcinoma in Children and Its Misdiagnosis:Report of One Case]. 3313 29