Gene/Protein Disease Symptom Drug Enzyme Compound
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Query: UMLS:C0038362 (stomatitis)
 8,852 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

The study consisted of 674 consecutive examinations done on patients on their first visit the College of Dentistry, King Saud University, Riyadh, over a 1-yr period starting January 1983. Men outnumbered women and 69% of the patients were Saudi Arabian nationals. Oral cancer was seen in one non-Saudi, whereas the overall prevalence of leukoplakia was 1.9%. The other lesions were leukoedema 8.3%, nicotinic stomatitis 2.5%, and lichen planus 0.6%. Except for lichen planus, all the lesions were almost exclusively found in men with a close relationship to oral tobacco habits. Leukoedema, nicotinic stomatitis and leukoplakia showed a tendency to occur together in varying combinations among smokers.
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PMID:Preliminary report on prevalence of oral cancer and precancerous lesions among dental patients in Saudi Arabia. 386 10

White lesions of the oral cavity are quite common and can have a variety of etiologies, both benign and malignant. Although the vast majority of publications focus on leukoplakia and other potentially malignant lesions, most oral lesions that appear white are benign. This review will focus exclusively on reactive white oral lesions. Included in the discussion are frictional keratoses, irritant contact stomatitis, and smokeless tobacco keratoses. Leukoedema and hereditary genodermatoses that may enter in the clinical differential diagnoses of frictional keratoses including white sponge nevus and hereditary benign intraepithelial dyskeratosis will be reviewed. Many products can result in contact stomatitis. Dentrifice-related stomatitis, contact reactions to amalgam and cinnamon can cause keratotic lesions. Each of these lesions have microscopic findings that can assist in patient management.
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PMID:Frictional Keratosis, Contact Keratosis and Smokeless Tobacco Keratosis: Features of Reactive White Lesions of the Oral Mucosa. 3067 62