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Query: UMLS:C0034065 (pulmonary embolism)
 14,979 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

A family is described in which the mother and three of seven children had atrial myxoma. The mother had biatrial myxoma; surgical treatment resulted in massive intraoperative embolization and death. Surgery was sucessful in two sons with left atrial myxoma and systemic arterial embolization. A third son had calcified right atrial myxoma with destruction of the tricuspid valve and episodes of syncope and pulmonary embolism; surgery including valve replacement, was successful. The mother's father and a brother had died suddenly without a definite diagnosis. The family data are consistent with dominant transmission. The possibility of finding affected relatives should be borne in mind when studying patients with atrial myxoma.
Am J Cardiol 1976 Aug
PMID:Atrial myxoma in a family. 13 7

Twenty-seven cases of ruptured chordae tendineae have been discovered during surgery for mitral regurgitation (9,3 %) : the highest incidence of ruptured chordae tendineae has been found among pure mitral insufficiency (36 %). In thirteen cases, the rupture was isolated, without any other valvular lesion. The syndrome described as characteristic of rupture was present in one third of our patients : isolated cases do not differ clinically from the others but for a more frequent acute evolution. In pure or predominant mitral regurgitation, surgery seems needed when clinical aggravation, acute or progressive, cannot be explained by arrhythmia, anaemia, pulmonary embolism, hyperthyroidism.
Acta Cardiol 1975
PMID:[Rupture of the mitral chordae tendinae]. 13 12

39 instances of mural thrombosis of the right side of the heart were observed among 2000 cases of post-mortem examinations. The right atrium was the most frequent site of thrombosis; the right ventricle was involved in 8 cases. The relationships between right sided thrombosis and rhythm disturbances, myocardial or valvular disease, myocardial infarction, pulmonary disease, neoplasm, sepsis and disturbance of coagulation are discussed. The high incidence of pulmonary embolism and their relationship with thrombosis of the right side of the heart are emphasized.
Acta Cardiol 1979
PMID:[Mural thromboses of the right heart. Clinico-pathological study]. 31 49

Serum-FDP was measured in 148 patients with AMI or pulmonary embolism within 48 hours of the onset of symptoms or within 48 hours of the admission to hospital. Within 48 hours of the hospitalisation 14.6% of the patients with AMI Rand 81.8% of the patients pulmonary embolism showed a serum-FDP elevation to over 10 units/ml. The use of the analysis is limited by the time which elapses until the result is available, by the short duration of the serum-FDP elevation and by the lack of specificity of the examination.
Acta Cardiol 1979
PMID:The value of serum fibrinogen degradation products in the differential diagnosis between acute myocardial infarction and pulmonary embolism. 31 86

The incidence of pulmonary perfusion defects after routine cardiac catheterization was assessed in 57 patients by comparing ventilation-perfusion lung scans obtained before and 1 day after catheterization. Patients were prospectively randomized to two groups, one in which right heart catheterization was performed using an antecubital venous cutdown procedure and one in which the percutaneous femoral vein approach was used. Seven patients (12 percent) had new postcatheterization perfusion defects consistent with pulmonary emboli. These patients did not differ significantly from patients without new defects in clinical characteristics, duration of catheterization, hemodynamic variables or route of right heart catheterization. The data suggest that pulmonary embolism may be a more common complication of routine cardiac catheterization than previously appreciated.
Am J Cardiol 1979 Mar
PMID:Incidence of new pulmonary perfusion defects after routine cardiac catheterization. 36 48

Anticoagulant-thrombolytic therapy and surgery, remain the topics of recent advances in the treatment of pulmonary embolism. The Authors have treated 3 patients with anticoagulant-thrombolytic therapy, and 14 patients with pulmonary embolectomy with cardiopulmonary bypass. By this experience, they suggest an indication for medical or surgical treatment for pulmonary embolism.
G Ital Cardiol 1979
PMID:[Massive pulmonary thromboembolism: medical or surgical treatment?]. 52 Jul 47

The Authors discuss their research on 800 endocardial permanent pacemakers (ventricular, atrial, and bifocal) implanted during six years. In the light of the examination of the literature, the AA. report the complications registered, in particular: lead's displacing 6.2%, run away 0.7%, marked hyperthermya 0.0%, haemorrage 0.4%, wound dehiscence 0.3%, asectic necrosis by decubitus 5%, septic necrosis 0.3%, perforation of the heart 0.2%, pulmonary embolism 0.1%. Both the most common causes of complications and the technical expedients employed to minimize the complication rates are being discussed.
G Ital Cardiol 1978
PMID:[Complications observed in 800 cases with implanted permanent endocardial pacemakers (author's transl)]. 75 65

In subjects carrying pacemakers, the catheter-electrode can induce, over a period of time, specific modifications at the level of the veins and the heart. The organism reacts to extraneous bodies with adhesive and sometimes thrombotic phenomena. The latter can occasionally be the cause of a pulmonary embolism (8 cases out of 105) or more rarely, the seat of a mycotic infection (one case). In the casuistry are included two cases of the complete perforation of the right ventricle (one of which was fatal) and four cases of partial perforation; in another subject a papillary muscle was perforated. Finally, one case of endocarditis was noted, the so called traumatic type, of the tricuspid valve.
G Ital Cardiol 1978
PMID:[Pathological observations on patients carrying pacemakers and clinical consequences. Reports of 105 observed cases (author's transl)]. 75 69

A 47 year old man with acute pulmonary embolism had severe pulsus paradoxicus in the absence of pericardial disease. Echocardiography demonstrated inspiratory failure of the aortic valve to open, a decreases in left ventricular cavity size and a decrease in mitral valve diastolic excursion.
Am J Cardiol 1977 Nov
PMID:Echocardiographic findings in severe paradoxical pulse due to pulmonary embolization. 92 Jun 17

A 39 year old pneumectomized patient presents a massive pulmonary embolism, dies within 3 hours and is supported inefficiently by cardiac massage with recurrent mydriasis during 2 hours. At that time, under extracorporeal cardiopulmonary bypass with a membrane oxygenator, the cardiac activity recovers immediatly due to right decompression and coronary perfusion. The patient is conscious within 5 hours. The cardiopulmonary bypass with a membrane oxygenator appears to be the best therapy when the cardiac massage fails to restitute a normal myocardial function. No embolectomy was performed. The patient died when the bypass was stopped after 48 hours. We conclude that the prolonged peripheral extracorporeal bypass followed by embolectomy is the best therapy of pulmonary embolism.
Acta Cardiol 1975
PMID:[Cardio-respiratory assistance with the extracorporeal membrane oxygenator for massive pulmonary embolism]. 108 51


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