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Query: UMLS:C0034063 (pulmonary edema)
 10,665 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

Eight cases of poisoning in workers cleaning silo are presented. Silo gas, produced during fermentation of vegetable material, contains very toxic nitrogen oxides. In this group three workers died within silo, four patients were hospitalized (one of them with acute toxic pulmonary oedema, two with sings of pneumonia, one had only transient decrease of consciousness) and recovered without detectable sequelae. One patient, in general good condition, refused hospitalization and recovered.
Pol Arch Med Wewn 1997 Jan
PMID:[Accidental poisoning with silo gas]. 923 52

The patients with chronic congestive heart failure and acute deterioration of heart failure (pulmonary oedema, significant reduction of blood pressure) have decrease liver's perfusion with signs of acute damage of liver's cells--ischemic hepatitis. Aspat, AIAT and LDH in blood rich very high level. The level of bilirubin, alkaline phosphatase and glucose increase slightly. Hepatotoxic viruses are never observed. The authors described a case of 34 years old man, who two years earlier had large myocardial infarction with aneurysm of heart and congestive heart failure. He was admitted to hospital in shock. The shock was caused probably by overdose of nitroglycerin. In ECG and Echo examinations he had no signs of acute myocardial infarction, but we observed serious damage of liver's cells with very high levels of AspAT, AIAT and LDH. Based on clinical and biochemical examinations we diagnosed ischemic hepatitis. The patient's clinical and biochemical tests were normalized after improvement of heart failure. Biopsy of liver was normal at that time. Prognosis in ischemic hepatitis depends on course of heart failure.
Pol Merkur Lekarski 1997 Dec
PMID:[Ischemic hepatitis]. 952 68

Acute myocardial infarction, a rare peripartum event, is accompanied by significant maternal and fetal mortality. We report a 41-year-old woman who developed an acute myocardial infarction during the third trimester of pregnancy. The case of infarction was complicated with pulmonary oedema on the 3 day after delivery. Coronary angiography was performed 7 weeks post partum and no coronary lesions were detected. Therapeutic options in such condition are discussed and review of relevant literature is presented.
Pol Arch Med Wewn 2002 Aug
PMID:[Acute myocardial infarction during pregnancy complicated with pulmonary oedema]. 1247 98

Cardiac abnormalities has been receiving increased attention in patients with systemic lupus erythematosus (SLE). Cardiovascular system involvement has been found to have a substantial effect on mortality and morbidity in patients with SLE [1]. Recent diagnostic methods using echocardiography examination have allowed the delineation of cardiac manifestations such as myocarditis and myocardial dysfunction, valvular disease, pericardial disease or pulmonary hypertension. A report of two cases is presented: 23-year-old man with acute myocarditis with left ventricular failure and pulmonary oedema as a initial presentation of active SLE, and 51-year-old woman with SLE, antiphospholipid antibodies, with history of cerebral embolic infarction, TIA and venous thrombosis and with mitral valvular dysfunction in course of nonbacterial thrombotic endocarditis. Pulmonary hypertension has been recognised in both patients probably as a result of vasculaopathy and intimal proliferation, vasculitis, thromboembolic disease or parenchymal lung disease in SLE. Recent advances in diagnosis and treatment have substantially improved the prognosis of patients with systemic lupus erythematosus and cardiovascular system involvement [2].
Pol Arch Med Wewn 2003 Feb
PMID:[Cardiovascular involvement in systemic lupus erythematosus: report of two cases]. 1287 81

Essential hypertension accounts for 95% of all cases of hypertension. A small number of patients (between 2% and 5%) have a reversible disease as the cause for raised blood pressure. Unilateral and bilateral renal artery stenosis may be responsible for secondary hypertension. Diagnosis and treatment of renal artery stenosis are of a great importance. Revascularization of ischemic kidney may correct blood pressure control and preserve renal function. Much data suggest close pathophysiological relation between renal artery stenosis, ischemic nephropathy and development of hypertension. However, it should be stressed that not every renal artery stenosis leads to hypertension and ischemic nephropathy. Therefore diagnosis of renal artery stenosis in hypertensive patient is not always equivalent with renovascular hypertension. The true prevalence of renal artery stenosis is unknown. In unselected population it accounts for less than 1% of hypertensive patients. Renovascular etiology of hypertension may be suggested by abrupt onset of hypertension, resistant and malignant hypertension or recurrent pulmonary edema of unknown etiology. Physical examination may reveal bruits over major vessels, including the abdominal aorta and renal arteries. The principle aim of the renal artery stenosis investigation is to confirm presence and size of vessel obstruction and its association with hypertension. Typical evaluation is based on imaging techniques and physiological studies. Former include: doppler duplex ultrasonography, conventional angiography, intraarterial and intravenous digital subtraction angiography, computed axial tomography, magnetic resonance angiography and intravascular ultrasonography. Functional studies are occasionally used. These are renal scintigraphy, evaluation of plasma renin activity in renal veins and evaluation of plasma rennin activity after ACE inhibition. Treatment of patients with renal artery stenosis and hypertension should restore vessel patency and inhibit its occlusion. Revascularization should elicit an improvement in or normalization of blood pressure control and renal function. Therapeutic approach include percutaneous renal artery angioplasty (PTRA), with or without stenting, revascularization by surgery and pharmacotherapy. PTRA is currently the first choice option. In general, it is simpler and similarly effective as surgical reconstruction. In some cases PTRA is completed with stent placement. It prevents immediate recoil but does not completely eliminate restenosis of revascularized artery. Surgical bypass is currently reserved for patients in whom PTRA and stenting fail and in patients with extensive atherosclerotic lesions. Patients with renal artery stenosis and hypertension should be provided with pharmacological treatment according to current recommendations. Specific procedures to limit associated risk factors of atherosclerosis should also be introduced.
Pol Merkur Lekarski 2003 Oct
PMID:[Renovascular hypertension: is it only the top of the iceberg?]. 1497 69

Increasing development of noninvasive methods of the cardiovascular system assessment as a consequence of increasing experience and technological progress cause more increasing aviability and interest of this techniques from the side of clinicians and scientists. The aim of this study was to present two noninvasive techniques: photoplethysmography and impedance cardiography with explanations of the basis of action, possibilities and limitations. Employment of this two methods opening new perspectives in haemodynamic assessment in many clinical situations for instance: shock, pulmonary edema, heart insufficiency, arterial hypertension and eclampsia. These methods could optimize farmacotherapy or stimulation parameters in patients with heart stimulation as well as to come to know and understand correlations between autonomic nervous system activity and haemodynamic changes. Noninvasivity and low costs of these procedures cause that impedance cardiography and photoplethysmographic blood pressure measurement will become wide use and acceptable methods all over the world.
Pol Merkur Lekarski 2005 Nov
PMID:[New noninvasive methods of cardiovascular system function assessment]. 1649 12

A case of a 70 year old female with hypertrophic obstructive cardiomyopathy who underwent alcohol ablation of the interventricular septum, is presented. Following the procedure, the pressure gradient decreased from 120 mmHg to 80 mmHg. However, 30 minutes after ablation the patients developed hypotension and pulmonary oedema. Echocardiography revealed a significant systolic anterior movement of the anterior leaflet of the mitral valve and elevated pressure gradient up to 200 mmHg. The patient underwent urgent surgery (myectomy), followed by the mitral valve replacement with a favourable outcome.
Kardiol Pol 2006 Sep
PMID:[Alcohol ablation of the interventricular septum in a patient with hypertrophic obstructive cardiomyopathy complicated by an increase in the pressure gradient in the left ventricular outflow tract. A case report]. 1705 31

A case of a 35-year-old female who developed acute myocardial infarction and pulmonary oedema during pregnancy, is presented. Coronary angiography revealed a critical stenosis of the left circumflex coronary artery and a successful angioplasty was performed. The course of a subsequent 13 year follow-up was uneventful.
Kardiol Pol 2007 Apr
PMID:[Long-term follow-up of a 35-year-old woman after myocardial infarction during pregnancy complicated by a pulmonary oedema - case report]. 1753 May 62

We present a case of severe complication of myocardial infarction -- acute mitral regurgitation caused by papillary muscle rupture. A 69-year-old man was admitted with chest pain lasting 1 hour and pulmonary oedema. ECG revealed ST-segment depression in leads II, III, aVF, V2-V6. Soon after admission the patient experienced respiratory disorders and consequently arrest. The patient was transferred in shock to the Department of Cardiothoracic Surgery, where he underwent successful artificial mitral valve implantation. One year later the patient is in good condition (NYHA class I) and the valve is fully functional.
Kardiol Pol 2007 Sep
PMID:[Acute mitral regurgitation caused by infarction-related papillary muscle rupture with successful surgical treatment: case report]. 1797 62

Acute lung injury (ALI), including acute respiratory distress syndrome (ARDS), is a critical condition consisting of acute hypoxaemic respiratory failure with bilateral pulmonary infiltrates of non-cardiogenic origin. Occasionally it develops in patients with acute or subacute central nervous system pathologies, such as increased intracranial pressure and brain stem lesions. The exact epidemiology of neurogenic pulmonary oedema is unknown. However, due to non-specific clinical manifestation it often remains undiagnosed. Its pathogenesis probably involves overactivation of the sympathetic autonomic system with pulmonary hypertension and increased vessel permeability. We present a case of successfully managed ARDS in a previously healthy patient with newly diagnosed and symptomatic haemangioblastoma of the medulla oblongata. In this context we also review current knowledge on the aetiopathogenesis, diagnostic criteria and treatment for neurogenic ARDS.
Neurol Neurochir Pol
PMID:Acute respiratory distress syndrome as a manifestation of brain stem tumour--a case report. 2005 60


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