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Query: UMLS:C0034063 (pulmonary edema)
 10,665 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

The authors report a personal case of mitral incompetence, due to rupture of the chordae tendineae and note the signs of this disease. The patient first developed pulmonary oedema with thoracic pain, a mitral systolic murmur and a presystolic gallop rhythm, which suggested the diagnosis in a patient in sinus rhythm with a normal size heart, the left atrium was expansive and there were obvious signs of pulmonary congestion. The rapidly fatal course may be explained by the large number of ruptured chordae tendineae. Autopsy showed that the mitral valve was normal in texture. Histological study of the ruptured cord, showed lesions of dense hyaline fibrosis and mucoid infiltration of the basic substance.
Sem Hop 1975 Oct 23
PMID:[Isolated rupture of the chordae tendineae of the mitral valve]. 17 95

The authors report 6 cases of acute respiratory failure complicating chronic bronchial and lung disease admitted to hospital with the diagnosis of: heart disease, 3 cases, pulmonary oedema, pulmonary embolism, atrial flutter; status asthmaticus : one case; neuro-psychiatric disease : 2 cases (toxic coma and agitation). The authors emphasize the frequency of chronic bronchial disease and recall the signs of acute decompensation discussing the possible difficulties in diagnosis and the therapeutic implications.
Sem Hop 1977 Sep
PMID:[Deceptive and revealing clinical forms of acute respiratory insufficience in chronic bronchopneumopathies]. 19 94

Non cardiogenic pulmonary edema is caused by an increase of alveolocapillary permeability, due to different etiologies: fat embolism, multiple trauma, septic shock, influenza pneumonia, aspiration syndrome... Chest radiographs exhibit interstitial and/or alveolar pattern, severity of injury is assessed by the magnitude of intra-pulmonary shunting. Pulmonary wedge pressure is normal, and increased pulmonary vascular resistance is sometimes evidence in prolonged evolutions, especially in fatal cases. Treatment consists in the suppression of hypervolemia, and ventilation with positive and expiratory pressure (PEEP). Extra-corporeal membrane lung oxygenation remains since now rather unsuccessful.
Sem Hop
PMID:[Non cardiogenic pulmonary edema (author's transl)]. 22 Jul 23

Acute pulmonary edema is a frequent complication of carbon monoxide poisoning. The hemodynamic study of a case with major hypoxemia revealed the cardiac origin of the acute respiratory failure. Treatment associated diuretics, cardiotonic drugs and a positive end expiratory pressure. This treatment did not however prevent the occurrence of a transient anterolateral injury on EKG. The pathophysiology of pulmonary edema in carbon monoxide poisoning is discussed and evidence in favor of it's hemodynamic origin are exposed.
Sem Hop 1983 Feb 03
PMID:[Acute pulmonary edema of hemodynamic origin in carbon monoxide poisoning]. 630 64

Long acting nitrate derivatives have varying hemodynamic effects of a piridoxilate-pentaerithrityle tetranitrate administration. The purpose of this study was to assess the hemodynamic effects of a piridoxilate-pentaerithrityle tetranitrate compound and measure their duration of action. The study was carried out in 11 patients with left ventricular incompetence. All other medications, except for anticoagulants, had been discontinued 5 days earlier and patients fasted during the investigations. Each patient was given the active product (100 mg pentaerithrityle tetranitrate) and the placebo orally, under double blind conditions. Pulmonary vascular pressure, peripheral arterial pressure, heart rate and cardiac output were measured over 8 hours for 2 consecutive days. The results show a decrease in the left ventricular preload from the 30th minute to the eighth hour, which is statistically significant from the first to the fourth hour. No changes were recorded in the postload or in any of the other parameters measured or derived. These findings suggest that the compound has an anti-anginal action by diminishing the pressure in the ventricular wall and reverses pulmonary edema in left ventricular failure. The long duration of action (at least 8 hours) allows prolonged dosage intervals.
Sem Hop 1983 Mar 10
PMID:[Prolonged hemodynamic effects of pentaerythrityl tetranitrate combined with piridoxylate]. 630 89

Acute lymphoblastic leukemia (cytologically typical) was diagnosed in an 18-year-old man presenting with major eosinophilia. Prednisone therapy was initiated but sudden congestive heart failure occurred (left ventricular insufficiency with pulmonary edema). Concomitantly, blood counts became normal. Remission of the leukemic process was obtained while severe mitral regurgitation developed, due to bacterial endocarditis. Successful mitral valvuloplasty was achieved. Remission is still persisting one year after diagnosis.
Sem Hop 1983 Jun 02
PMID:[Acute lymphoblastic leukemia with major eosinophilia. Inaugural cardiac insufficiency, endocarditis grafting and valve replacement]. 630 90

In 200 young patients with apparently idiopathic spontaneous pneumothorax, the following radiologic features were analyzed: degree of collapse on the initial chest film, areas of atelectasis, and presence of blebs, apical opacities, fibrous adhesions, pleural effusions, and controlateral shift of mediastinal structures. Confrontation of apical changes with pathologic findings in operative specimens suggests that mesothelial rupture with reactive hyperplasia results in a "pneumatization chamber" visible as a bullous image. Following drainage, homolateral shifts of mediastinum and four cases of pulmonary edema were recorded. Risk factors for pulmonary edema include severe pulmonary collapse with areas of atelectasis, persisting for more than 48 hours and an aspiration which either exceeded 1.5 l. of air or was performed with a depression of more than 30 cm of water.
Sem Hop 1984 Mar 08
PMID:[Radiology of spontaneous pneumothorax in young patients. Apropos of 200 cases]. 632 55