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 11,717 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

In a 55-year-old woman patient with congenital Horner's syndrome the ptosis and miosis disappeared completely for a short time after a bout of coughing. Pharmacologically the defect was localized in the second neuron. No etiology or pathogenetic mechanism of the phenomenon could be identified.
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PMID:[Horner's syndrome reversible by coughing]. 336 89

This study aimed to determine the extraretinal effects of melatonin upon the eyes of an avian species, the House Finch (Carpodacus mexicanus). Twelve birds (full-grown, second-year males) each received a Silastic tubing intraperitoneal implant, six containing melatonin (average release = 24 micrograms/d/bird; = M birds) and six being empty (= C birds). Microscopic study of pupillary and palpebral behaviors during the final week demonstrated lesser pupillary diameters and interpalpebral distances in M birds under all test conditions. These effects could have diminished mean light levels reaching parts of the retina. Characteristics of the relative miosis and ptosis of M birds resemble signs in some CNS disorders, such as altered inhibition of the Edinger-Westphal nucleus, and especially lesions in, or lowered activity of, higher sympathetic centers (a subtype of Horner's syndrome). Weights of eyes and their parts were the same in M and C birds, contrasting with previously reported results from male Golden Hamsters, possibly due to species differences and/or preexperimental attainment of full growth in the finches. Effects of melatonin on pupillary and palpebral behaviors, demonstrated here for the first time, foster caveats for simplistic experimental designs and interpretations with melatonin when sensory-neural-behavioral interactions are affected. Quantitative changes in pupillary and palpebral behaviors may, nevertheless, provide a window for monitoring central actions of melatonin in living test subjects in chronic studies.
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PMID:Effects of melatonin implants on structures and behaviors of the house finch (Carpodacus mexicanus) eye. 372 27

It is well known that Horner's syndrome is characterized by ptosis, miosis, and facial anhydrosis. However, the congenital form of Horner's syndrome is relatively uncommon, and it is scarcely known among otolaryngologists. In this report, two cases of congenital Horner's syndrome are described, both encountered recently in the recent author's practice. In the first case, the patient was seeking unnecessary medical treatment for a facial abnormality, not for his eyes. Thermography performed on this patient confirmed his complaint. Having had the first experience made diagnosis in the second case easier. Thus, although rare, this syndrome may be encountered in an otolaryngologist's daily practice and it is important to recognize its symptoms. It is suggested that some suffering from this disease may go undetected due to physicians' lack of familiarity with it.
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PMID:Congenital Horner's syndrome in otolaryngologic practice. 381 84

Horner's Syndrome (ptosis, anisocoria, and anhydrosis) developed in a 22-year-old woman. A mediastinal mass was noted on chest x-ray. Further evaluation led to a diagnosis of Hodgkin's disease, nodular sclerosis type. Horner's syndrome is an unusual initial manifestation of Hodgkin's disease, and, in this case, it was due to oculosympathetic damage from mediastinal compression. Because the potential for cure is high in Hodgkin's disease, this diagnosis should be considered in patients presenting with Horner's syndrome.
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PMID:Horner's syndrome: an unusual presentation of Hodgkin's disease. 404 77

We report a 22-year-old male who underwent a left thoracostomy and left lower lobectomy for a shotgun wound of the left anterior chest. During surgery, two chest tubes were placed in the left hemithorax at the level of the first and second intervertebral spaces. On the 10th postoperative day, the patient developed a left-sided miosis and ptosis. The diagnosis of a left preganglionic Horner's syndrome was confirmed by pharmacologic testing. The Horner's syndrome was unchanged 3 months after discharge. We conclude that the chest tubes damaged the preganglionic sympathetic fibers of the left orbit resulting in this patient's Horner's syndrome.
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PMID:Iatrogenic Horner's syndrome secondary to chest tube thoracostomy. 622 21

Horner's syndrome developed in 2 horses after routine jugular venipuncture. Signs included unilateral sweating of the face in both horses and ptosis in 1 horse. The signs resolved within 14 hours. Signs of a perivascular injection did not develop in either horse. Although Horner's syndrome has been reported after perivascular jugular injections these cases illustrate that the syndrome may develop following routine intravenous injections.
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PMID:Transient Horner's syndrome following routine intravenous injections in two horses. 649 May 12

Fifteen patients with Horner's syndrome have been examined for the presence of upside down ptosis, and this symptom could be demonstrated in 10 of the patients. Miosis occurred in all patients, and ptosis of the upper lid was seen in 14 patients. These were the symptoms which originally enabled the diagnosis to be made. Enophthalmus was present in only 4 patients. The anatomic basis is briefly discussed, and it is advocated from the easy demonstration of upside down ptosis, together with its independence of voluntary compensation by the patient, that it should be looked for as a routine when examining for Horner's syndrome.
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PMID:Upside down ptosis in patients with Horner's syndrome. 665 3

A patient with Stage I systemic sarcoid (affecting the mediastinum and cervical lymph nodes) is presented. This patient had ptosis of the eyelid and miosis (Horner's syndrome) due to involvement of the cervical-thoracic sympathetic nerves by the granulomatous process. Bony destruction simulating periodontal disease occurred in the molar-premolar region and caused pain and paresthesia. Systemic sarcoid may occur more commonly in the jaws than reported cases would indicate. Sarcoidosis should be considered in the differential diagnosis when periodontal disease appears refractory to local therapy.
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PMID:Systemic sarcoidosis presenting with Horner's syndrome and mandibular paresthesia. 695 36

Horner syndrome (ptosis, miosis, anhidrosis, and facial and conjunctival vasodilation) is a recognized complication of lumbar epidural analgesia for labor and delivery. Alone, it presents no significant risk to mother or fetus, as resolution is spontaneous and complete. Horner syndrome may, however, be associated with significant maternal hypotension and therefore should be an indication for close maternal and fetal monitoring to provide reassurance.
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PMID:Horner syndrome during lumbar epidural analgesia for obstetrics. 708 23

Raeder's syndrome consists of ipsilateral ptosis, miosis and facial pain with intact facial sweating. When not associated with other neurologic signs, the clinical course of this conditions is self-limited. Patients will have resolution of facial pain but persistence of miosis and ptosis. Treatment is symptomatic with arteriography reserved for those patients with protracted symptoms or atypical presentations. A case of Raeder's paratrigeminal syndrome is presented with abnormal dilation of the subcavernous portion of the internal carotid artery thought to be secondary to inflammation of the adjacent sphenoid sinus. Facial pain and the abnormal dilation of the carotid artery resolved, but miosis and ptosis persisted. Because of the therapeutic indication and prognostic value, an awareness of Raeder's syndrome is stressed when evaluating patients with facial pain or possible Horner's syndrome.
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PMID:Raeder's syndrome associated with internal carotid artery dilation and sinusitis. 713 15


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