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Query: UMLS:C0030552 (paresis)
 5,831 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

The association of left vocal cord paresis with an esophageal diverticulum pointing to the left at the aorto-bronchial level constitutes a syndrome resulting from mediastinal fibrosis in that region. Demonstration of this type of diverticulum in a case of unexplained left cord paresis establishes mediastinal fibrosis as its cause.
Am J Roentgenol Radium Ther Nucl Med 1975 Sep
PMID:A new symdrome of left vocal cord paresis and esophageal diverticulum due to mediastinal fibrosis. 81 67

The distal non-traumatic ulnar nerve compression syndrome can be dividded into two types: -i) the proximal (paralytic) type caused by compression in the "Loge de Guyon", with involvement of both the superficial sensory and the deep branch, including the motor branch to the hypothenar muscles, and, ii) the distal, more common, purely motor type (the deep ulnar branch syndrome) with paresis of the interossei and the adductor pollicis, and less commonly of the hypothenar muscles. Whereas the proximal type has various causes, particularly pressure and occupational trauma, the distal type is almost exclusively the result of extra-neural ganglion cysts. An accurate diagnosis is made possible by electromyography and nerve conduction studies. The sensory nerve action potentials, distal motor latency to the hypothenar and adductor pollicis muscles, and the corresponding muscle action potentials after nerve stimulation are pathologically altered, according to the type of lesion. Although spontaneous recovery may occur, operation is the treatment of choice, provided that simple pressure palsy is eliminated.
Neurochirurgia (Stuttg) 1977 Sep
PMID:[Distal ulnar nerve compression at the wrist. "Loge de Guyon" and "deep ulnar branch" syndrome (author's transl)]. 90 63

When 2 horses were dosed with cultures of a Fusarium moniliforme isolate that had previously caused only hepatosis, 1 developed brain oedema and hepatosis, and the other only leukoencephalomalacia. A 3rd horse developed both leukoencephalomalacia and hepatosis after being dosed with another isolate obtained from maize which was associated with a natural outbreak of the nervous form of the disease. Since leukoencephalomalacia and hepatosis could be induced by the same culture material, it was concluded that both syndromes were manifestations of the same toxicosis. There was also some evidence that leukoencephalomalacia might be specifically induced by the administration of smaller doses of the culture material to horses over a longer period. The clinical signs of nervous disorder included ataxia, paresis, apathy, hypersensitivity, frenzy, and other locomotory and psychic disturbances. Autopsy showed that the brains were oedematous, and focal areas of liquefactive necrosis were present in the cerebral white matter. In 1 case the malacic areas were not confined to the subcortical white matter but were microscopically visible in the cerebral cortex as well. An histopathological examination of the areas bordering on the malacic areas revealed rarefication of the white matter, perivascular haemorrhages, oedema and cellular infiltration composed mainly of plasma cells and eosinophiles. Many of the macrophages in these areas contained lipfuscin-like granules, but these granules also occurred extracellularly in the neuropil. In the layers of the cortex nearest the malacic areas, satellitosis and neurophagia were commonly seen.
Onderstepoort J Vet Res 1976 Sep
PMID:Leukoencephalomalacia: a mycotoxicosis of Equidae caused by Fusarium moniliforme Sheldon. 101 50

During February 1975, a tremorgenic neurotoxicosis decimated a herd of cattle in the northern Transvaal. This hitherto unidentified disease was characterized by hypersensitivity, incoordination, a peculiar stiff-legged gait of the hind legs, severe generalized tumors of the sketetal muscles, progressive paresis, paralysis and constipation. The most notable gross pathological lesions were degenerative and necrotic changes in certain skeletal muscles, haemorrhages on the serosal surfaces, especially on the dorsal aspect of the rumen, and gastro-intestinal stasis. Microscopical examination of the central nervous system revealed cytopathological changes consisting of degeneration and necrosis of the large motor cells in the ventral horns of the spinal cord and bigger neurones in numerous nuclei of the medulla oblongata, midbrain and thalamus. By feeding the suspect ration and its component parts to cattle and sheep, it was possible to identify mouldy sorghum beer residue as the toxic component in the ration. A. clavatus, the dominant fungus on the toxic residue, was readily isolated in pure culture. The entire syndrome was then reproduced in a yearling Friesland steer dosed with pure cultures of the A. clavatus isolate grown on autoclaved non-toxic sorghum beer residue. The toxic principle is not known, but it does not appear to be patulin, tryptoquivalone, tryptoquivaline, or any other known tremorgen.
Onderstepoort J Vet Res 1976 Sep
PMID:A highly fatal tremorgenic mycotoxicosis of cattle caused by Aspergillus clavatus. 101 54

A case report of Moebius syndrome is presented. The syndrome includes bilateral facial and abducens nerve paresis, muscular hypoplasia, adactylia, club foot, and possible mental retardation. This particular patient had bilateral facial and abducens nerve paralysis, mandibular hypoplasia, pectoral muscle hypoplasia, adactylia, and bilateral clubbed feet.
J Oral Surg 1975 Sep
PMID:Moebius syndrome. 105 90

Those who live in areas where Malaria is endemic, acquire immunity by continuous contact. This immunity cannot be acquired during a short holiday. Children in endemic areas acquire a more severe form of malaria during the period of developing immunity and more often suffer complications like acute hemolytic anemia and, in the case of plasmodium falciparum infection, cerebral malaria. This is a report of 39 cases of cerebral malaria which corresponds to an acute encephslopathy with high temperatures, generalized tonic-clonic spasms and unconsciousness. All children were between 6 months and 5 years old. Cerebral malaria at higher ages is rarely seen in Malawi. But its frequency depends on the intensity of endemic infection and the geographic distribution of the types of malaria. 11 (29%) of the 39 children died. Treatment was with chloroquine against which there was no resistance in East Africa for falciparum infections and with plasmaexpanders. In 1 case permanent neurologic changes a spastic cerebral paresis, were seen. Unconsciousness lasting more than 36 hours appears to be a bad prognostic sign. The CSF is clear and normal except for an occasional rise in protein never higher than 90 mg%.
Klin Padiatr 1975 Sep
PMID:[Malaria in children, with special reference to cerebral malaria (author's transl)]. 110 Aug 97

In nine cases of botulism B infection due to food poisoning acute onset of accommodation paresis, mydriasis, and dry-eye symptoms were the prominent clinical findings. Impairment of salivary secretion as a further effect upon cholinergic autonomic innervation was detectable for months. Guanidine treatment had a beneficial effect, whereas administration of antitoxin in the late and benign form was not effective.
Am J Ophthalmol 1975 Sep
PMID:Ocular involvement in benign botulism B. 116 91

Cluster headache is frequently characterized by pain localized to the orbital area. There is often associated ipsilateral oculosympathetic paresis with varying degrees of blepharoptosis and miosis. The ophthalmologist is often confronted with such cases; however, the atypical presentations and the subtle clinical findings may obscure the diagnosis. As cluster headache is a benign condition, accurate recognition is essential to spare the patient potentially harmful diagnostic studies.
Ann Ophthalmol 1975 Sep
PMID:Ophthalmic presentations of cluster headache. 116 26

Following the North American example, mal-practice suits become more common in Germany. The rare complication of a paresis of the femoral nerve following vaginal hysterectomy is reported. Among 1307 vaginal hysterectomies, including 33 radical vaginal hysterectomies between 1969 and 1974, three cases occurred. In one case, the surgeon was sued but the patient lost the mal-practice suit. In another case a diminished employability was recognized later. The causes and the prevention of damage of the femoral nerve during vaginal procedures are discussed.
Geburtshilfe Frauenheilkd 1975 Sep
PMID:[Paresis of the femoral nerve following vaginal hysterectomy and its medico-legal implication (author's transl)]. 117 11

This is a case report of a newborn infant with an isolated complet paresis of the peroneal nerve, diagnosed shortly after birth. The etiology of this quite rare observation in a newborn is discussed and the literature reviewed.
Monatsschr Kinderheilkd 1975 Sep
PMID:[Isolated peroneal nerve palsy in a newborn (author's transl)]. 117 54


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