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Query: UMLS:C0030552 (paresis)
5,831 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

Remitting paresis of the left leg accompanied by left trigeminal neuralgia led to the diagnosis of multiple sclerosis in a 46-year-old woman. Over the following 6 years, an incomplete syndrome of the spinal cord developed along with bilateral trigeminal pain. Neuroradiological and neurosurgical exploration a neurinoma located ventrolaterally at C1/C2 on the left side. It is emphasized that since trigeminal fibres descend as far as the upper part of the C2 segment, trigeminal neuralgia should not be considered as an exclusively supraspinal symptom.
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PMID:High cervical neurinoma (C1/C2) diagnosed falsely as multiple sclerosis because of trigeminal neuralgia. 49 15

Three cases of histologically verified neurinomas of the oculomotor nerve are reported. The preoperative diagnosis in all three cases was not made; on the contrary all cases were diagnosed as sphenoid ridge meningiomas. The oculomotor neurinoma manifests itself in the beginning with a discrete progressive palsy of the oculomotor nerve leading in later stages to complete ophthalmoplegia. Proceeding the oculomotor nerve paresis or going parallel to it is a functional loss of the homolateral optic nerve sometimes progressing to amaurosis. Unilateral exophthalmos as well as frontal or orbital neuralgic pain with or without sensory disorders in the area of trigeminus-I are characteristic for the clinical picture in later stages, all symptoms characteristic for the syndrome of the apex orbitae, resp. the superior orbital fissure, resp. the anterior cavernous sinus. The differential diagnosis has to consider above all the sphenoid ridge meningioma, the trigeminal neurinoma and the numerous tumors within the cavernous sinus (aneurysmas, meningiomas, chondromas, metastases of carcinomas, pituitary adenomas etc.) or the middle cranial fossa. Plain X-ray, carotid angiogram and computer tomogram are essential diagnostic means for localization and extension of the tumor but not for histological diagnosis. Oculomotor neurinomas are very seldom. The three observed cases did not have any relation to a possible generalized neurofibromatosis.
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PMID:[Neurinoma of the oculomotor nerve (author's transl)]. 65 Dec 44

A 38-year-old man developing slowly progressive left facial paresis was admitted to our hospital. The clinical diagnosis of "adhesive arachnoiditis was made. The first operation was performed in December 1987, and his symptom disappeared postoperatively. Three years later, left facial paresis recurred together with trunkal ataxia. A computed tomography and magnetic resonance image revealed two tumors located at the left cerebellopontine angle region and in the left middle cerebral fossa. These two tumors were thought to arise in the different cranial nerve. Under the clinical diagnosis of acoustic neurinoma associated with facial nerve neurinoma, the two step-operation was designed for total removal of the tumor in December 1990 and January 1991. Intraoperative finding confirmed that these two tumors had the different origin. Pathologic diagnosis was compatible with neurinoma. This patient had no family history of "Neurofibromatosis".
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PMID:[Acoustic neurinoma associated with facial neurinoma--a case report]. 128 93

A 38-year-old man was referred by his general practitioner to our department on 28 October 1991, with a 2-week history of vertigo. A left aural polyp was identified. The audiogram showed a moderate conductive loss on the left side. Computed tomography (CT) and magnetic resonance imaging (MRI) confirmed the presence of the expanding lesion in the descending portion of the facial nerve. However, there was no seventh nerve paresis. At operation, the neurinoma (Schwannoma) filled the middle ear cleft and extended from the genu to the stylomastoid foramen. The floor of the middle ear had been eroded, exposing the jugular bulb. Facial nerve paresis is the usual presenting feature of a facial neurinoma. The case is presented for the reason that the absence of facial palsy as a presenting feature is rather rare, especially in the cases with large tumor and extensive bone erosion.
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PMID:Intratemporal facial nerve neurinoma without facial paralysis. 129 96

A rare case of trochlear nerve neurinoma is described. Including this case, the number of reported intracranial tumors arising from the sheaths of the third, fourth, and sixth cranial nerves is 38. By site and relationship to the nerve segment, they fall into three groups: cisternal, cisternocavernous, and cavernous. In cisternal tumors of the third and sixth nerves, paresis of the nerve hosting the tumor is the unique nerve deficit; by contrast, in those of the fourth nerve, paresis of the trochlear nerve can be absent and that of the third nerve present. In the latter tumors, a peculiar ataxic hemiparesis syndrome is produced by midbrain compression. Cisternocavernous neurinomas often cause symptoms of intracranial hypertension, while cavernous neurinomas bring about two clinical features: paresis of one or more nerves of the cavernous sinus and a clinicoradiological orbital apex syndrome. At surgery, generally cisternal neurinomas are totally removed and the nerve source of the tumor identified; in cisternocavernous and cavernous neurinomas, total removal of tumor and identification of the parent nerve have been reported in only half of the cases. In the majority of parasellar neurinomas, clinical differences can be found between those arising from the nerves governing eye movement and those arising from the gasserian ganglion.
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PMID:Neurinoma of the third, fourth, and sixth cranial nerves: a survey and report of a new fourth nerve case. 144 Feb 7

Only 10 cases of oculomotor nerve neurinoma have been reported previously. The authors report the rarity of a neurinoma originating from Schwann cells of the oculomotor nerve. The diagnosis was based on the initial sign of oculomotor nerve paresis without involvement of other cranial nerves, neuroradiological and surgical findings, and histological features of the specimen obtained at surgery.
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PMID:Neurinoma of the oculomotor nerve--case report. 169 35

The results obtained from a complete neuro-otological test battery were examined statistically in order to select measurement variables which would optimally indicate significant differences between four groups: normal patients, patients with partially compensated unilateral peripheral vestibular deficit, patients with an acoustic neurinoma and patients with central (brainstem) vestibular deficit. A stepwise-discriminant analysis was performed on measurements of slow-phase velocity obtained from each test. The primary measurements selected to assign a subject optimally to one population were the canal paresis (CP) of the caloric test, the eye-tracking gain contralateral to the deficit for a 15 deg/s stimulus, the gain asymmetry for optokinetic nystagmus with a 30 deg/s stimulus, and the level of spontaneous nystagmus. The resulting classifications were 100% correct for normal and central deficit patients. However, the division between peripheral deficit and acoustic neurinoma patients overlapped causing about 30% false classifications of neurinoma patients: some 20% of the peripheral deficit patients were classified as normal. If the CP was not available the discriminant analysis substituted the rotating chair response for 5 deg/s2, in place of CP. This substitution caused a 10 to 20% decrease in classification accuracy.
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PMID:Classification of peripheral and central (pontine infarction) vestibular deficits. Selection of a neuro-otological test battery using discriminant analysis. 201 53

A 64-year-old man with right aural discharge presented complaining of progressive deafness. Other otological symptoms were absent and specifically there was no seventh nerve paresis. A right aural polyp was identified and biopsied. Histology showed the polyp to be a schwannoma. Subsequent temporal bone computed tomography showed expansion of the distal facial canal. At operation, the schwannoma filled the middle ear cleft and extended from the genu to the region of the stylomastoid foramen. The floor of the middle ear had been eroded, exposing the jugular bulb. Facial paresis is the usual presenting feature of a facial schwannoma, while deafness, ageusia and reduced lacrimation are variable, dependent upon the site of the lesion. The absence of facial palsy as a presenting feature is very rare and this case illustrates the need for histological examination of all abnormal aural material.
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PMID:Facial schwannoma without facial paralysis. 258 62

A case of neurinoma of the trochlear nerve presenting with the sudden onset of headache followed by transient paresis of the right trochlear nerve in a 37-year-old woman is reported. Unique clinical manifestations of the tumor are discussed with a brief review of five cases reported in the literature.
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PMID:Trochlear neurinoma. 311 1

A case of intracranial hypoglossal neurinoma is reported. A 32-year-old man with a history of unsteady gait and headache for one and a half years was admitted to our hospital on September 23, 1983. Neurological findings on admission were nystagmus toward the left, left IXth and Xth cranial nerve paresis, left cerebellar signs, paresthesia of the left upper and lower extremities on neck flexion, and left pyramidal signs. Atrophy or fasciculation of the tongue was not noted. Computed tomography demonstrated a large isodensity and partially low density mass in the posterior fossa which was markedly enhanced and sharply margined with contrast medium. The tumor extended 45 mm over the edge of the foramen magnum. The maximum size was 45 mm X 40 mm. The coronal and sagittal reconstruction CT scan clearly demonstrated the attachment and extension of the tumor. Enlargement of the hypoglossal canal was clarified by changing the window level, window width and using 2 mm slice CT scan. Anterior-posterior x-ray view of the skull tomography showed enlargement of the hypoglossal canal, as indicated by the CT scan. The left hypoglossal canal was 7 mm and right was 3 mm in diameter. Vertebral angiography demonstrated that the tumor was located in the left posterior fossa, but no tumor stain appeared. This patient did not have XIIth cranial nerve palsy, but the neuroradiological findings strongly suggested a hypoglossal neurinoma, especially the findings of the CT scan and the anterior-posterior x-ray view of the skull tomography. On October 5, suboccipital craniotomy with Cl laminectomy was performed in prone position.(ABSTRACT TRUNCATED AT 250 WORDS)
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PMID:[A case of intracranial hypoglossal neurinoma]. 380 8


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