Gene/Protein Disease Symptom Drug Enzyme Compound
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Query: UMLS:C0030552 (paresis)
 5,831 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

We experienced three female patients with sudden sensorineural hearing loss. All the patients showed elevation of anticardiolipin antibody in the serum. Immunoglobulin (Ig) M anticardiolipin antibody was present in case 1 (a 34-year old woman). IgG anticardiolipin antibodies were present in cases 2 (a 50-year-old woman) and 3 (a 9 year old girl). The patient in case 1 showed hearing disturbance of low tone in the left ear and normal vestibular function in both ears. The case 2 patient had an average hearing level of 81.7dB at 0.5. 1, and 2kHz and severe canal paresis in the right ear. In case 3 the patient showed an average hearing level of 53.3dB in the right ear and normal vestibular function in both ears. It was suggested that thrombosis which might have been induced by anticardiolipin antibody could have caused the sudden sensorineural hearing loss in these patients. Steroid therapy cured the hearing disturbance completely in case 1. Steroid and prostaglandin E1 therapy did not improve the hearing disturbance in case 2. Steroid, prostaglandin E1, and ticlopidine hydrochloride therapy improved the hearing disturbance in case 3. It appears that not only steroids but also prostaglandin E1 and ticlopidine hydrochloride therapy may be effective in patients with sensorineural hearing loss associated with anticardiolipin antibody.
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PMID:[Sudden sensorineural hearing loss associated with anticardiolipin antibody]. 891 11

A 65-year-old man was suffering from recurrent manic psychosis accompanied by weight loss. He also had a history of pleural effusion, aspecific migratory non-deforming seronegative polyarthritis, sensorineural hearing loss and semicircular canal paresis. Whipple's disease (WD) had been diagnosed at the age of 63 years. On admission to hospital) he had weight loss, diarrhoea in combination with an organic brain syndrome, hemiparesis and ophthalmoplegia, including internuclear ophthalmoplegia (INO). A clinical diagnosis of central nervous system (CNS) WD was made. MRI revealed a thalamus lesion that halved in size during sulfamethoxazole-trimethoprim treatment. The organic brain syndrome and ophthalmoplegia diminished also, as did the cerebrospinal fluid (CSF) IgG level. A review of CNS WD is presented and implications for treatment are discussed.
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PMID:Whipple's disease and the central nervous system. A case report and a review of the literature. 893 Apr 20

We studied the labyrinthine function in a group of 72 children aged between 4 and 14 years affected by unilateral sensorineural hearing loss of probable viral origin. From the analysis of the results obtained we confirm the concomitance of cochlear and vestibular damage. However, there were no statistically significant differences between type of audiogram at onset of hearing loss and type of electronystagmography (ENG), while we found a direct correlation between the presence of vertigo or dizziness and type of ENG. Finally hearing recovery was influenced by the presence of vertigo or labyrinthine function alterations. The results of statistical analysis confirmed a significant statistical difference between patients with vertigo or dizziness (V(+)) and those without vertigo (V(-)) and also between patients with ENG 3 (subjects with spontaneous nystagmus or positional nystagmus and canal paresis ipsilateral to the cochlear lesion) and those with ENG 1 (subjects without spontaneous nystagmus or positional nystagmus and with normal vestibular reflex). In fact, hearing recovery was worse in V(+) group and in ENG 3 group.
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PMID:Electronystagmography findings in child unilateral sensorineural hearing loss of probable viral origin. 946 27

Kernicterus is a preventable life-long neurologic syndrome caused by severe and untreated hyperbilirubinemia during the neonatal period. High levels of bilirubin are toxic to the developing newborn. In full-term infants, hyperbilirubinemia symptoms include severe jaundice, lethargy, and poorfeeding. Features of kernicterus may include choreoathetoid cerebral palsy, mental retardation, sensorineural hearing loss, and gaze paresis. Kernicterus is not a reportable condition in the United States, and its prevalence is unknown; however, a pilot registry at a Pennsylvania hospital documented 90 cases in 21 states from 1984 to June 2001 (L. Johnson, Pennsylvania Hospital, Philadelphia, personal communication, 2001). This report summarizes case histories of four full-term, healthy infants who developed kernicterus and underscores that to prevent kernicterus, newborns must be screened and promptly treated for hyperbilirubinemia.
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PMID:Kernicterus in full-term infants--United States, 1994-1998. 1142 29

A 25-year-old female developed permanent, fluctuating sensorineural hearing loss (SNHL), disabling vertigo, and tinnitus following an uneventful spinal anesthesia for cesarean section. At her first visit to the ear-nose-throat (ENT) department approximately 2 months postoperatively, pure-tone thresholds revealed profound SNHL on the right side whereas thresholds were within normal limits on the left side. The recruitment score (SISI) was 95% at 2000 Hz on the right side. Directional preponderance towards the right and the right canal paresis were evidenced by bithermal caloric testing. At follow ups the pure tone thresholds have shown some improvement, but fluctuating SNHL, disabling vertigo attacks, and tinnitus have remained. These findings imply a cochlear pathology causing endolymphatic hydrops possibly induced by lumbar puncture for spinal anesthesia.
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PMID:Permanent sensorineural hearing loss following spinal anesthesia. 1236 13

We report the recovery of the inferior vestibular neural system disorder by monitoring the vestibular-evoked myogenic potential (VEMP) in two patients suffering from acute sensorineural hearing loss with vertigo. Patients presented absent VEMP, canal paresis, and severe hearing disorder. After 10 months, the function of the inferior vestibular neural system was recovered in both cases while that of the superior vestibular neural system was recovered in one case and partially recovered in the other. In addition, the hearing sensitivity was only partially recovered in both cases. The recovery of the inferior vestibular neural system was confirmed by our current results. These results suggested that time course of recoveries of the superior and inferior vestibular neural systems, and of the cochlear neural system, were somewhat different in the two cases.
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PMID:Recovery of vestibular-evoked myogenic potential: relationship to other neural disorders in two patients with acute sensorineural hearing loss. 1241 78

Cavernous angiomas of the internal auditory canal are rare lesions. The authors present a case of a 29-year-old lady with multiple infratentorial cavernous angiomas, whose sister had previously undergone surgery for a similar supratentorial lesion. She initially presented with an acute brainstem haematoma, secondary to a pontine cavernous angioma. Three years later she developed progressive right-sided sensorineural hearing loss and facial nerve paresis due to an internal auditory canal lesion. This was removed via the translabyrinthine approach and was found to be a cavernous angioma. This report underlines the multiple and dynamic nature of familial cavernous angiomas, as well as the importance of follow up to determine whether new symptoms are due to the enlargement of known angiomas or the development of new ones. As far as the authors are aware, this is the first report describing a cavernous angioma of the internal auditory canal in the context of familial and multiple infratentorial angiomas.
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PMID:Cavernous angioma of the internal auditory canal. 1516 13

Sound-induced vestibular evoked myogenic potentials (VEMP) on the surface of the sternocleidomastoid muscle constitute the basis of the saccular function test. In order to assess the saccular function, VEMP test was performed in a patient having unilateral Meniere's disease with profound sensorineural hearing loss in the contralateral ear. The saccular response was absent on the affected side with a normal caloric response. However, VEMP response was normal in the contralateral ear with profound sensorineural hearing loss and canal paresis. There may be saccular dysfunction in Meniere's disease.
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PMID:[Clinical value of vestibular evoked myogenic potentials in saccular dysfunction in Meniere's disease: a case report]. 1605 99

A 51-year-old man developed sudden vertigo, right hearing loss and dysphagia. Examination revealed right Horner syndrome, spontaneous torsional-horizontal nystagmus, right central type facial palsy, dysarthria, reduced soft palate elevation without gag reflex, left hypesthesia, right dysmetria and imbalance. Audiometry and bithermal caloric tests documented right sensorineural hearing loss and canal paresis. Brain MRI and cerebral angiography documented right lateral medullary infarction from vertebral artery dissection, without involvement of other parts of the brainstem supplied by the anterior inferior cerebellar artery (AICA). This case suggests artery-to-artery embolism as a possible mechanism of isolated vertigo or hearing loss from labyrinthine infarction.
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PMID:Embolic internal auditory artery infarction from vertebral artery dissection. 1658 Jun 95

Osteomas occur throughout the temporal bone and, depending on their location, may cause tinnitus, hearing loss, vertigo, and facial nerve paresis. We present a rare case of a 25-year-old woman with a mastoid osteoma enlarging over a 6-month period. Other than a cosmetic deformity of her upper neck, the patient was asymptomatic. After surgical removal of the bony neoplasm, the patient was noted to have a high-frequency sensorineural hearing loss. This case study presents clinical, radiologic, intraoperative, pathologic, and audiometric findings of a mastoid osteoma and a review of the literature.
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PMID:Giant mastoid osteoma with postoperative high-frequency sensorineural hearing loss. 1835 40


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