Gene/Protein Disease Symptom Drug Enzyme Compound
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Query: UMLS:C0030552 (paresis)
 5,831 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

Six cases with CT scan evidence of paramedian thalamic lacunar infarcts in the region of the paramedian thalamic arteries (4 unilateral left and 2 bilateral) are described. The main acute symptoms included disorders of consciousness, hypersomnia and sometimes vertical gaze paresis. An amnesic syndrome of variable degree and duration was observed in all cases, and in 1 of them was still present 2 years after the onset. In addition, we report a case of lateral thalamic infarction, which showed a predominantly aphasic (transcortical sensory type) clinical picture.
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PMID:Lacunar thalamic infarcts and amnesia. 261 24

We reported a case of amnestic syndrome caused by a hematoma in the left thalamus. The case was that of a 68-year-old, right-handed man who suddenly showed amnestic syndrome. He had neither motor paresis nor sensory disturbance. Clinical examination showed he had disorientation, anterograde amnesia and mild retrograde amnesia. Immediate recall and remote memory were intact, but recent memory was severely impaired. CT scan revealed a high density area in the anterior medial part of the left thalamus. We concluded that amnesia in this patient was caused by fasciculus mamillothalamicus damage because of a hematoma during thalamic hemorrhage.
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PMID:[A case of amnesic syndrome caused by hematoma in the left anterior medial thalamus]. 855 65

We report here a case of amnestic syndrome with executive dysfunction and attention deficit caused by cerebral infarction in the right temporal lobe was reported. An 80-year-old, right-handed female with complaints of memory impairment, was admitted to our hospital. She had neither motor paresis nor sensory disturbances. She exhibited disorientation, retrograde amnesia, anterograde amnesia, executive dysfunction and attention deficit. Although her immediate recall and remote memory were almost intact, her recent memory was moderately impaired. Both verbal memory and non-verbal memory were impaired. Brain MRI revealed cerebral infarction in the right temporal lobe involving the parahippocampal gyrus, and SPECT demonstrated low perfusion areas in both the cerebral hemispheres involving basal ganglia and the right thalamus. After a 4-month rehabilitation program, her memory impairment improved considerably. We consider that the reason that contributed to these improvement include the fact that her lesion was unilateral and that her executive dysfunction and attention deficit also improved. This finding suggests that memory is related to attention and executive function.
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PMID:[Amnestic syndrome caused by cerebral infarction in the right medial temporal lobe--case report]. 1788 82