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Query: UMLS:C0024523 (malabsorption)
7,319 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

Enlargement of the valvulae conniventes is an integral part of the pattern diagnosis of primary small bowel disease causing malabsorption. The pathophysiology underlying enlargement of the fold and the most typical diseases with prominent folds leading to malabsorption are discussed. Differential diagnosis is based on enlargement of the fold and on secondary signs.
AJR Am J Roentgenol 1976 Sep
PMID:Pathophysiology of enlargement of the small bowel fold. 6 Aug 85

A personal series of 28 cases of malabsorption with villopathy (22 Gee, 1 Whipple, 1 alpha-chain, 1 hypogammaglobulinaemia, 3 not yet diagnosed) is presented. The response to a gluten-free diet is the essential clue to diagnosis. Where sensitivity is not found, diagnosis becomes difficult and requires careful immunological, histopathological and parasitological investigation, which is not always conclusive. The main features of the series are indicated, along with the criteria employed in the diagnosis of some unusual forms.
Minerva Med 1976 Sep 19
PMID:[28 cases of malabsorption syndromes with villopathy]. 6 68

Non-specific ileojejunitis, characterised by mild to moderate structural changes in the intestinal mucosa and often associated with asymptomatic malabsorption, is known to occur in the tropics and in countries with hot climates. This acquired condition is probably related to environmental factors, and some consider it to be a subclinical form of tropical sprue. Changes in the intestinal mucosa typical of non-specific ileojejunitis were found in 10 indigenous Indians as well as 5 people of Latin stock living in the Southern Peruvian sierra, where tropical sprue has not as yet been demonstrated and which has a temperate climate.
Lancet 1978 Sep 09
PMID:Non-specific ileojejunitis in Arequipa, Peru. 7 18

Currently, 6 agents of viral gastroenteritis with properties similar to the Norwalk agent have been described. These are the Hawaii, Montgomery County, W, Ditchling, and Cockle agents, as well as the Norwalk agent. These agents are 25 to 27 nm in diameter, are nonenveloped, and have a buoyant density of 1.38 to 1.40 g/cc in CsCl. Multiple antigenic types exist among these agents. These particles have been detected by immune electron microscopy in feces of acutely ill patients. None has been successfully cultivated in vitro, and suitable animal models of disease do not exist. Studies in volunteers indicate that acute infection is associated with a reversible histopathologic change in the jejunal mucosa and with transient malabsorption. Pathogenesis of these changes remains unknown. Other similar agents will likely emerge from ongoing studies.
J Am Vet Med Assoc 1978 Sep 01
PMID:Norwalk agent-like particles associated with gastroenteritis in human beings. 10 Apr 81

A total of 121 children from an orphange were studied. Their ages ranged from 2 to 10 years and represented different ethnic backgrounds. The prevalence of malnutrition was 45% and their gastrointestinal symptomatology in response to graded amounts of milk intake was evaluated. None of the children complained when milk intakes were of 200 cc; 10% complained of gastrointestinal discomfort and/or diarrhea when 300 cc of milk (equivalent to 12 g of lactose) were administered, and 54% presented lactose malabsorption unrelated to ethnic background, sex or malnutrition. It is concluded that the prevalence of milk intolerance is minimal when milk is given in the usual amounts.
Arch Latinoam Nutr 1979 Sep
PMID:[Milk intolerance in Colombian children, its prevalence and relation to lactose malabsorption]. 12 Jul 6

Hypolipidemias can be divided in primary, familial and hereditary forms and symptomatic forms which may accompany other diseases. The primary hypolipidemias (abetalipoproteinemia, hypobetalipoproteinemia and analphalipoproteinemia) are very rare. Severe hypolipidemia can be found in some peoples (e.g. the Masai). This article is chiefly devoted to secondary hypolipidemias such as those associated with malabsorption, malnutrition and maldigestion including protein-losing gastroenteropathy, with liver diseases, endocrine diseases (hyperthyroidism, hirsutism) and anemia. Finally, the hypolipidemias secondary to the formation of autoantibodies against HDL and LDL in M-gradient, carcinoma and rheumatoid arthritis are briefly reviewed.
Schweiz Med Wochenschr 1975 Sep 27
PMID:[Hypolipidemias]. 17 Jun 75

A series of 29 cases of amyloidosis of the alimentary tract is reported. Five cases (17%) were primary amyloidosis; 14 cases (48%) were amyloidosis secondary to other diseases (such as chronic inflammatory and neoplastic diseases); 10 cases (35%) were amyloidosis of the heredo-familial type connected with Familial Mediterranean Fever. In 23 patients (79%) the diagnosis was established by biopsies, and in 6 more cases on autopsy. Gastrointestinal involvement was found in all age groups. Gastro-enterologic complications observed in the present series include: diarrhea, malabsorption, ileus and gastrointestinal bleeding. In addition other conditions such as jaundice (3 cases), esophagitis and acute hemorrhagic pancreatitis were observed. In 22 patients proteinuria was observed and in 13 patients the nephrotic syndrome. Among 17 patients, in 11 the clinical picture before death was that of terminal renal failure. The survival after diagnosis among 14 patients reached 4 years in 9 cases, and 19 years in one case. The diagnostic value of the rectal biopsy is emphasized.
Nouv Presse Med 1976 Sep 25
PMID:[Gastrointestinal amyloidosis]. 18 89

A giant axonal neuropathy is described in an adult with no evidence of a childhood onset of his disease and no history of exposure to toxic hydrocarbons. A causal relation to vitamin B12 malabsorption is proposed. This case suggests the need to expand the spectrum of diagnoses that must be considered when giant axons are encountered in peripheral nerve biopsy specimens.
Acta Neuropathol 1977 Sep 26
PMID:Giant axonal neuropathy: possibly secondary to vitamin b12 malabsorption. 19 35

We studied five patients (two men and three women, age between 58 and 76 years) with clinical and electrophysiological signs of polyneuropathy. Routine neurological, hematological, and gastroenterological studies as well as procedures to test fat malabsorption were performed. Folate determinations were done using both radioactive and Lactobacillus casei methods. Two patients displayed the signs of subacute combined degeneration of the spinal cord with polyneuropathy, while three had only signs of neuropathy. All had low serum folate concentration, long-standing gastrointestinal disease, and deficient folate intake. The D-xylose absorption test gave values in all patients, while none displayed the classical malabsorption syndrome. The patients had substantial improvement or recovered (according to clinical and electrophysiological measurements) after periods ranging from 9 to 39 months of folate therapy. Such acquired folate-responsive polyneuropathy has two principal characteristics: mixed sensorimotor with mainly sensory deficits, and involvement of one or both of the lower extremities much more extensively than the upper extremities.
Arch Neurol 1978 Sep
PMID:Polyneuropathy and folate deficiency. 21 Jul 47

Long-standing complete obstruction of the superior mesenteric artery and vein and recurrent gastrointestinal bleeding from varices within the bowel were encountered in a child as a complication of midgut malrotation and volvulus. This condition is unusual, but should be considered in cases of gastrointestinal bleeding, or when small bowel biopsy is contemplated for the evaluation of diarrhea or malabsorption in cases of malrotation.
Gastroenterology 1979 Sep
PMID:Mesenteric vascular occlusion and varices complicating midgut malrotation. 31 54


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