Gene/Protein Disease Symptom Drug Enzyme Compound
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Query: UMLS:C0020639 (hypoproteinemia)
 1,134 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

The paper deals with 8 cases of different forms of bacterial respiratory infections (bronchiectasis, pleuropneumonia, tuberculosis), in which humoral immune deficits were noted: 2 dysgammaglobulinemia with IgA absence, 3 hypogammaglobulinemia with IgG deficit as well as IgA and IgM absence, 2 hypogammaglobulinemia with IgG and IgM deficit and IgA absence, and 1 hypogammaglobulinemia with IgA absence and IgM deficit. Stress is laid on the indications given by the antibody titer research, the pleural exudate hypoproteinemia being also added to those already known. A correct chemotherapy associated with the substitution treatment proved to by efficient.
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PMID:[The characteristics of bacterial respiratory infections in patients with humoral immunodeficiencies]. 134 11

A 77-year-old woman was hospitalized repeatly due to frequent hemoptysis and production of bloodly sputum for several years. Bronchography in 1989 revealed bronchiectasis. She had complained of abdominal pain and diarrhea since 1991, and her urine was first positive for protein in 1992. She was admitted to our hospital in October 1992 because of edema, anemia, and hypoproteinemia. Despite treatment, renal dysfunction and the gastrointestinal disorder progressed and she died in January 1993. An autopsy revealed diffuse depositions of amyloid in many organs, especially in the kidney and the gastrointestinal tract. This amyloid protein was identified as AA protein, which was suggestive of secondary amyloidosis. Bronchiectasis appears to have been the disease underlying this patient's amyloidosis.
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PMID:[Amyloidosis secondary to bronchiectasis]. 875 22

A 64 years old woman without systemic immunological disorders was admitted to our hospital because of a productive cough, low grade fever and bloody sputum. Chest X-ray revealed multiple nodules with calcification, infiltrates and bronchiectasis. Laboratory findings showed mild hypoproteinemia and elevated sedimentation rate. Both Nocardia farcinica and Mycobacterium intracellulare were isolated from the bronchial lavage fluid. Administration of sulfamethoxazole-trimethoprim improved her symptoms. In a recurrent study of bronchial lavage N. farcinica was not isolated, but M. intracellurale was still isolated. We believe that N. farcinica may cause infectious exacerbation of chronic lung disease: non-tuberculous mycobacteriosis and bronchiectasis.
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PMID:[A case of pulmonary Nocardia farcinica infection in a patient with non-tuberculous mycobacteriosis]. 1078 84