Gene/Protein Disease Symptom Drug Enzyme Compound
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Query: UMLS:C0018991 (hemiplegia)
 3,997 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

Sixteen cases of spontaneous dissection of the cervical internal carotid artery (6 verified) are described. The mean age was 45 years. The clinical picture varied from simply headache and a bruit to hemiplegia and aphasia. Eleven patients had transient ischemic attacks. Headache, facial pain, a subjective bruit, oculo-sympathetic palsy and transient monocular blindness were present in various combinations in two-thirds of cases and their presence suggested the correct diagnosis. Examples of suspected dissection of the intracranial internal carotid, middle cerebral, posterior cerebral and extracranial vertebral arteries are also presented. Spontaneous dissection is more common than the literature indicates.
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PMID:Spontaneous dissection of cervico-cerebral arteries. 64 2

The symptom of burning orofacial pain may help to identify the site of ischaemia in otherwise pure motor strokes resulting from infarction of the ventral pons. A patient with hemiplegia due to ventral pontine infarction, in whom burning oral and mid-facial pain was a prominent initial symptom, is described. Similar pain preceded transient episodes of the 'locked-in' state. Awareness of this herald symptom may permit early recognition and careful monitoring of patients at risk of progressing to the 'locked-in' state.
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PMID:Burning oral and mid-facial pain in ventral pontine infarction. 237 75

Preoperative embolization for intracranial meningioma has been performed at many institutions and its effectiveness has been well recognized. The complications of embolization such as facial pain, fever and facial nerve palsy, et al, were mild and temporary except embolus migration into intracranial vessels, but a peritumoral hemorrhage due to preoperative embolization was extremely rare. Recently we have experienced such an unusual complication, then we describe this complication here and discuss the mechanism of hemorrhage briefly. A 73-year-old female who had the left falx meningioma underwent preoperative embolization with gelfoam powder through the transfemoral route. About 10 hours later, she developed disturbance of consciousness and right hemiplegia. At that time, CT scan showed peritumoral hemorrhage and an increase in midline shift. An emergency craniotomy was performed and total removal of the falx meningioma (Simpson grade II) and evacuation of the hematoma were done. The postoperative course was uneventful. We conclude that gelfoam powder (average particle size 40-60 mu) is a useful material for preoperative embolization but may at times cause peritumoral hemorrhage.
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PMID:[Preoperative embolization with gelfoam powder for intracranial meningioma causing unusual peritumoral hemorrhage--with reference to the mechanism of hemorrhage]. 382 72

Sudden hearing loss is common, but unexplained in many cases. Although usually attributed to a viral infection of the inner ear in most patients, the abrupt onset of the hearing loss in many patients argues against a viral etiology. We present 13 cases of unexplained sudden hearing loss who meet the diagnostic criteria for migraine. All had the sudden onset of hearing loss and other neurologic phenomena that could be attributed to vasospasm, including vertigo, amaurosis fugax, hemiplegia, facial pain, chest pain, and visual aura. We suggest that vasospasm of the cochlear vasculature was the cause of the sudden hearing loss in these patients. A personal and family history of migraine should be sought in patients with sudden hearing loss and when found, a trial of antispasmodic agents should be considered.
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PMID:Migraine as a cause of sudden hearing loss. 866 32

The authors present a sample of five patients, 3 women and 2 men, with history of the dislocation of the mandible. The period ranged between 7 days and 7 months and all the patients looked for medical treatment, but did not have a correct diagnostic. The initial diagnostic was tetanus, sequela for VCA, food intoxication. The younger patient was 23 years old and the older was 63 years old. The first male patient was 23 years old and his first complaint was facial pain, rigidity, difficulty in speaking, chewing and the diagnosis received was Tetan, after 7 days. The second female patient was 61 years old and her first complaint was difficulty in speaking and chewing. The diagnosis was food intoxication after 7 month. The third patient was 54 years old. The first complaint was idiopathic, after 1 month. He did'nt managed to shut up his mouth--diagnosis idiopathic cause. The fourth patient was 63 years old and his first complaint was assimetric and facial pain due a neurological remains, after 4 month. And finally the fifth patient. He was 56 years old and the first complaint was an intense difficulty in speaking and shut out his mouth--hemiplegia in the left face due neurological remain, after 3 month. The authors make a review about the criteria for a diagnosis of dislocation of mandible and the benefits of immediate treatment.
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PMID:[Acute facial pain associated with mandibular motion damage: considerations on a clinical sample]. 1043 42

Mucormycosis is a life-threatening fungal infection that occurs in immunocompromised patients. The most common predisposing risk factor for mucormycosis is diabetes mellitus. Rhino-orbito-cerebral mucormycosis is the most common form in diabetic patients and is characterized by paranasal sinusitis, ophthalmoplegia with blindness, and unilateral proptosis with cellulitis, facial pain with swelling, headache, fever, rhinitis, granular or purulent nasal discharge, nasal ulceration, epistaxis, hemiplegia or stroke, and decreased mental function. Diabetic ketoacidosis is the most common and serious acute complication of diabetic patients. We herein report 2 cases of fatal rhino-orbito-cerebral mucormycosis in a patient with diabetic ketoacidosis.
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PMID:Rhino-orbito-cerebral mucormycosis in patients with diabetic ketoacidosis. 2462 43

We report the case of a 66-year-old female with hemiplegia cruciata and severe facial pain due to infarction of the cervicomedullary junction. She presented to the hospital with complaints of acute-onset left facial pain and gait disturbance. Neurological examination revealed narrow left palpebral fissure, severe left facial pain and hypothermoesthesia, weakness predominantly in the left upper and right lower extremities, decreased pain and temperature sensation in the right lower extremity, decreased vibration sensation in the left lower extremity, hyperreflexia in the left upper extremity, and mild ataxia in the left upper and lower extremities. Brain MRI revealed a high-intensity lesion in the left cervicomedullary junction on diffusion-weighted and fluid-attenuated inversion recovery images. Hemiplegia cruciata due to the pyramidal tract injury at the cervicomedullary junction is an uncommon clinical manifestation. However, in patients with hemiplegia cruciata, identifying the lesion location may be difficult. Clinicians should consider the possibility of pyramidal decussation lesions. Anatomical differences, in the course of pyramidal tract fibers between the upper and lower limbs have been considered in the pyramidal decussation. Hemiplegia cruciata in this case was primarily caused by the impairment of the left upper limb pyramidal fibers after the pyramidal decussation and the right lower limb pyramidal fibers before the pyramidal decussation.
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PMID:[Hemiplegia cruciata and severe facial pain due to infarction of the cervicomedullary junction: a case report]. 3289 44