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56,091 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

Hemorrhage from an intrinsic vascular malformation of the optic chiasm (chiasmal apoplexy) is an uncommon cause of sudden visual loss with chiasmal visual field defects. This paper describes one case of sudden visual loss with an anterior chiasmal visual field defect due to rupture of an intrachiasmatic venous angioma and contrasts it with the clinical presentation of a case of hemorrhage from an intrachiasmatic arteriovenous malformation causing severe headache and a less apoplectic onset of visual symptoms. The variable clinical presentation and the accuracy of high resolution post-contrast cranial CT scan in the diagnosis of intrachiasmal hemorrhage is highlighted.
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PMID:Chiasmal apoplexy due to intrachiasmatic vascular malformation rupture. 343 24

This is a case report of intracranial multiple cavernous angioma occurred in a member of a lineage of familial cavernous angioma. This lineage had manifested multiple small angiomata in the extremities on seven members in her family. The patient, somewhat retarded 19-year-old woman, was admitted to Fukuoka University Hospital because of convulsive seizure and headache occurred several months before admission. She had a large head and her IQ was 71. Bilateral mild hearing disturbance and a vascular anomaly in the left eyeground were noted. There were multiple hypodermic nodules on four extremities. EEG showed spike and wave focus in the right parietal region with irregular background. Plain skull X-ray disclosed scattered small calcifications. CT scan revealed multiple high density areas in the bilateral cerebral hemispheres and splenium and cerebral angiograms presented avascular mass in the right parietal region. The tumor in the right parietal lobe which seemed to be epileptogenic focus and the hypodermic nodules of the upper extremities were removed. Pathological findings of intracranial mass and hypodermic nodule were similar, these were cavernous angioma. The mother and a younger brother of the patient had similar hypodermic nodules on the extremities, and brother's nodule was removed and confirmed cavernous angioma. Eight families of familial cavernous angioma were described in the literature. This presented case is a peculiar one on the view-point of occurrence in the central nervous system among the familial hypodermic manifestation on the extremities and coexistence of retinal vascular anomaly.
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PMID:[Multiple and familial cavernous angiomas in the brain and extremities--case report]. 370 45

It has already been noted that hemangioblastoma is occasionally complicated with various diseases, especially retinal angioma, cysts of kidney and/or pancreas, vascular disorders, and furthermore about 10% of hemangioblastoma are multiple. The authors report here a case of multiple cerebellar hemangioblastomas accompanied with congenital deafness, juvenile diabetes mellitus and retinal angioma. The patient, a 38-year-old man who complained of disturbance of consciousness, headache and vomiting, admitted to our hospital on October 6, 1982. He was born in consanguineous marriage family and his elder sister was also suffered from congenital deafness and juvenile diabetes mellitus, but no angiomas. CT and angiography showed a left side cystic and a right side small cerebellar lesions with remarkable ventricular enlargement. Ventriculo-peritoneal shunt and suboccipital craniectomy & removal of the tumors were performed on October 6, and November 8, 1982 succeedingly. From a point of view of hereditary occurrence, the authors investigated these complicated lesions recognized in this patient. It revealed that this case did not belong to any other categories of hereditary syndromes which were already reported and so authors considered that this was an extremely rare condition. Moreover, in case of multiple hemangiomas, cerebral angiography could be a more useful method than CT scan to detect multiplicity of the lesions in the posterior fossa.
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PMID:[A case of multiple cerebellar hemangioblastomas with congenital deafness, juvenile diabetes mellitus and retinal angioma]. 370 51

We reviewed the clinical and CT findings in six patients with mesencephalic hematomas. All patients were normotensive. Headache and vomiting preceded impairment of consciousness. In all patients, unequal pupils reacted poorly to light but retained the near response. Upgaze was impaired in all patients. CT showed a unilateral nonenhancing midbrain hematoma in all cases and intraventricular blood in five. Four-vessel angiography in all cases showed no neoplasm, angioma, or aneurysm. All patients survived with persistent pupillary abnormalities and restriction of upward gaze.
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PMID:Mesencephalic hemorrhages: clinical and computed tomographic correlations. 370 72

A case of a cavernous angioma of the cauda equina is presented. The patient was a 28-year-old man who experienced sudden low-back pain and headache without neurological symptoms. Lumbar puncture revealed subarachnoid hemorrhage. He had suffered a similar episode 3 years previously. Selective spinal angiography did not demonstrate any abnormal vascularity. Metrizamide myelography and magnetic resonance imaging were useful in demonstrating the presence of a tumor. Laminectomy at L1-3 and total removal of the tumor were performed without neurological deficit.
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PMID:Cavernous angioma of the cauda equina producing subarachnoid hemorrhage. Case report. 378 46

The presentation of 129 cases of cerebral angioma is reviewed. There were 4 main methods of presentation: haemorrhage, epilepsy, deteriorating neurological signs and headache. The fifth common method of presentation, heart failure, almost exclusively confined to infants, was not seen in the current series which includes no paediatric cases. Standard techniques of investigation principly angiography and CT scanning were used in diagnosis and the current indication for surgery was haemorrhage. The mortality rate per haemorrhage in the series considered in natural terms was 3.6%, neurological deficit as a result of the haemorrhage, either major or minor, occurred in a total of 8%, 4% each. Complete excision of the angioma was performed in 69 cases, although in 3 of these it was necessary to reoperate following postoperative angiography which was then used again to confirm total removal. There was 1 death in this series of radical removals, a mortality rate of 1.5%. Six cases however, showed significant neurological deterioration postoperatively, a major morbidity of 8.7%. From the period of observation of these cases and the instance of haemorrhage the annual bleeding rate would appear to be 4.2%. From follow-up of our cases where the lesion was not completely removed the re-bleeding rate would appear to be 2.6%.
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PMID:The management of cerebral arteriovenous malformations. 397 44

We have studied three patients with angiographically documented cerebellar venous angioma (CVA). One patient had a subacute cerebellar hematoma and underwent posterior fossa craniotomy for evacuation of the hematoma and excision of the malformation. A hemorrhagic venous infarction of the brain stem and cerebellum occurred, and the patient died three weeks postoperatively. A second patient with an unruptured CVA had a history of headaches, tinnitus, and vertigo. Conservative treatment was elected, and the patient's condition remains unchanged after 11 months of follow-up. The third patient, recently diagnosed as having an unruptured CVA had episodic vertigo and disequilibrium. Conservative treatment was chosen, and he is asymptomatic after six months of follow-up. Based on a review of 24 other cases of CVA plus our experience we could not conclude any definite trend regarding natural history or treatment. However, conservative treatment seems the logical choice in patients with unruptured CVA.
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PMID:Cerebellar venous angiomas. A continuing controversy. 398 13

8 cases of cavernous haemangiomas operated and histologically proven are presented. 5 patients (5/8) were admitted because of epileptic seizures, one patient (1/8) because of pure headache, another one because of a focal neurological defect and the last because of intracranial hypertension. Computed axial tomography is a sensitive procedure for detection of cavernomas. Of 7 cases examined by this technique, 7 (7/7) have shown a well circumscribed round or oval hyperdense nodule, with calcifications in 6/7 cases, a slight surrounding oedema in 3/7 cases and without mass effect, except if there is a visible hematoma at operation (2/8). After contrast administration, 5/7 malformations were enhanced and 2/7 displayed draining veins, a fact that we underline. The differential diagnosis includes a low grade calcified glioma, a thrombosed arterio-venous malformation, a venous angioma or an intracerebral hematoma. Surgical excision is the best treatment of these vascular malformations that bleed frequently, and improved all the patients in this series (8/8).
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PMID:[Intracerebral cavernous angiomas]. 408 98

A 27 year old man had attacks of violence for three years before he died in 1945. Necropsy revealed multiple cavernous angiomata of the brain. This man's daughter died in 1969 at the age of 28 years after a year of intermittent headaches. Necropsy showed a large cavernous angioma in the right temporal lobe. Until now the literature has contained only scanty circumstantial evidence for a familial occurrence of cavernous angiomata of the brain.
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PMID:Familial occurrence of cavernous angiomata of the brain. 553 7

The authors described a case of arteriovenous malformation (AVM) of the choroid plexus, and presented a review of literature. This 40-year-old male experienced a sudden onset of severe headache and vomiting on February 5th 1982. At the other hospital, CT scans revealed marked intraventricular hemorrhage, however his general condition was relatively good without a loss of consciousness, motor weakness and sensory disturbance. After about a month of conservative treatment, he was transferred to the Kochi Medical School Hospital. On admission, he had shown no neurological deficit except for slight occipital headache. Transfemoral cerebral angiography revealed an angioma of the choroid plexus, which was fed by the anterior and medial posterior choroidal arteries and drained into the internal cerebral vein. CT scans showed a small high density area due to the nidus of angioma at the interspace of bilateral frontal horns of the lateral ventricles. On March 25th 1982, using an anterior transcallosal approach, the angioma was totally removed. The histological diagnosis was AVM. The post-operative course was uneventful and the patient discharged without adding neurological deficit on April 12th 1982. Review of literature revealed 27 documented cases of angioma of the choroid plexus; 12 cases of AVM, 5 of cavernous angioma, 2 of telangiectasia, 1 of venous angioma and the other 7 of undefined description. Almost all cases were initiated with an episode of variable intracranial hemorrhage, particularly of intraventricular hemorrhage. Clinical course of them, however, were variable. On the contrast to male prevalence of AVM located in the other sites, the ratio of female to male was 2:1.(ABSTRACT TRUNCATED AT 250 WORDS)
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PMID:[Arteriovenous malformations of the choroid plexus--a case report]. 663 12


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