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Query: UMLS:C0018681 (headache)
56,091 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

Cluster headache is a defined disorder which is often mistaken in spite of its characteristic picture. The different types of cluster headache and their clinical symptoms are reviewed in detail. Predisposing factors, frequency of other medical disorders as well as personal and psychological characteristics are described. The treatment of the attack and the prophylaxis includes ergot alkaloids, methysergide, cortisone, lithium and oxygen. Some aspects of etiology and pathogenesis are discussed.
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PMID:[Cluster headache]. 711 33

The authors conducted periodic electroencephalographic (chrono-EEG) investigations in 64 subjects including 48 with various vasomotor headaches and 16 healthy ones. Chrono-EEG investigations carried out at different times of the day during wakefulness in healthy subjects and patients with Horton's (cluster) headaches showed lack of changes of the biochemical activity of the brain. In the group of patients with common vasomotor headaches the background activity was labile and evidence of decreased wakefulness was found in evening hours. In patients with migraine two patterns of chrono-EEG curves were elicited; normal an significantly activated at noon and during evening hours. The obtained results of these investigations seem to suggest a lack of uniformity of the pathomechanism of migraine. Further investigations have been started.
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PMID:[Chronoelectroencephalographic studies in migraine and related headaches]. 719 70

The skin of both temples was examined ultrastructurally in six patients with cluster headaches and in three controls. An increased number of mast cells were present in the patients irrespective of whether they were in a cluster period or in a quiescent phase. The mast cells were found perivascularly and in the vicinity of cutaneous nerves in the patients, whereas they were predominantly found in perivascular areas in controls. Mast cell degranulation was not more prominent on the side of the pain and occasional degranulated mast cells were found in controls. These findings are consistent with the hypothesis that cluster headache is due to an axonal reflex in the trigeminal system, initiated perhaps by latent viral infection of IgE activation of mast cells.
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PMID:Cluster headache. Ultrastructural aspects and pathogenetic mechanisms. 722 18

Thirty-one patients with Horton's headaches and a control group of 31 healthy subjects were investigated by means of the 16-factor personality Questionnaire of R. B. Cattell. The group of 31 patients were investigated additionally with the tests of Benton, Bender and the Intelligence Scale Wechsler-Bellevue. Personality questionnaires demonstrated in patients with Horton's headaches a fairly high intensity of factors G and Q4 (frustration of lower needs blocked by higher needs). No changes were found which could suggest presence of cerebral microlesions in the group with Horton's headaches. In the tests for intellectual efficiency the mean results pointed to a slightly above average general intelligence level in patients with Horton's neuralgia and reduced ability of memory, concentration and learning speed.
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PMID:[Selected psychological studies in Horton's headache]. 726 54

1. Cluster headache is a severe unilateral head or facial pain, which lasts for minutes or hours, commonly associated with ipsilateral lacrimation and blockade of the nostril. It usually recurres once or more daily for a period of weeks or months, separated by intervals of freedom. The sphenopalatine ganglion seems to play a very important role in its pathology, 2. We created a technic of alcohol infiltration of this ganglion through a supra-zygomatic way, based on the research of the maxillary nerve by neurostimulation and the bone contact with the pterygoid process. 3. We observed a relief of pain and parsympathetic disturbances in more than 85% of our 120 cases, with a follow up between 6 months and 4 years. 4. Results obtained are discussed and analysed in connection with the definition of cluster headache. (Acta anaesth. belg., 1981, 32, 101-107).
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PMID:Cluster headache and sphenopalatine block. 729 8

The syndrome of cluster headache variant is characterized by the occurrence of three combined symptoms: atypical cluster headaches, multiple jabs, and background vascular headaches. Atypical cluster headaches are localized headaches that occur several times daily, usually without any headache-free periods. They differ from the typical chronic cluster headache in their location, duration, frequent shifting, and frequency. Multiple jabs are short-lasting, sharp pains of variable severity and location. Background vascular headache is a chronic, continuous often unilateral headache of variable severity that throbs at rest or begins to throb during exertion. We have studied 54 patients between the ages of 14 and 78 years (average age, 40.5 years). Forty-five (83%) patients responded to indomethacin. Complete control was achieved in 50% of the patients. The nine patients who did not respond to indomethacin were depressed. These nine patients responded well to tricyclic antidepressants.
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PMID:Cluster headache variant. Spectrum of a new headache syndrome. 730 99

In 18 men with Horton's headaches the levels of the following hormones were determined in the serum: TSH, T3 and T4. The determinations were performed during disease relapse and before starting treatment. The control group included 18 blood donors. In the control group the mean TSH level was 3.97 +/- 0.88 microunits/ml, T4 level was 109.06 +/- 26.88 nmol/l, and T3 1.48 +/- 0.14 nmol/l. In the group of patients the mean values were: 2.58 +/- 2.09 microunits/ml, 97.62 +/- 21.81 nmol/l, 1.07 +/- 0.21 nmol/l respectively. The analysis of the results showed statistically significant differences in the concentrations of T3 between the compared groups. In the light of these results the authors believe that there is not sufficient support of the hypothesis on a hypothalamic pathogenesis of the disease. The causes of reduced T3 concentration in these patients are discussed.
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PMID:[Thyrotropic and thyroid hormone (T3 and T4) content in Horton' s headache patients]. 734

The authors analysed electroencephalographic findings in 36 patients with Horton's headache (cluster headaches). It was found that EEG findings in Horton's headache are usually normal and show considerable stability. Slight EEG changes observed in a low number of cases were usually similar to those observed in the general population. The analysis of bioelectric activity of the brain in patients with this headache suggests an extracerebral mechanism of this disease.
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PMID:[Analysis of electroencephalographic findings in patients with Horton's headache]. 737 97

13, mostly male, cases of cluster headache and 15, mostly female, cases of atypical facial neuralgia were submitted to the psychosomatic anamnestic interview of Seguin. A very high proportion of patients in both groups had suffered from early deprivation of physical gratification. Pain itself started initially after a sequence of masked depression followed by dental or facial intervention in most atypical facial neuralgias, and by emotional stress in half of the patients with cluster headaches. Pain afterwards still hid and expressed a current underlying depression in both groups.
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PMID:The onset of facial pain. A psychological study. 744 41

Prophylactic use of Lithium salts in patient suffering from cluster headaches has been evaluated looking at the mean number of headache attacks in one critical period and the mean weekly duration of the period itself. These two elements have been compared to those observed on other drugs treatments. Plasma Lithium monitoring has been performed weekly during the trial. Authors discuss the results reported and the hypothetic basis of them.
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PMID:[Lithium therapy in Horton's neuralgia: preliminary results]. 744 25


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