Gene/Protein Disease Symptom Drug Enzyme Compound
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Query: UMLS:C0012833 (dizziness)
 9,689 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

In patients with portal hypertension and tense ascites, large-volume paracentesis improves patient comfort and may improve systemic hemodynamics. However, it has been avoided in nonedematous patients because of concern for complications, including intravascular volume depletion. In this study, 12 nonedematous patients with chronic liver disease, portal hypertension and tense ascites underwent 14 large-volume (5-liter) paracenteses for the relief of discomfort and/or respiratory distress. Plasma volume was measured directly by a dilution method with 125I-labeled human serum albumin prior to and at 24 or 48 hr after 13 of the paracenteses. All patients felt better postparacentesis. No dizziness, hypotension, tachycardia, encephalopathy or change in mean serum sodium, creatinine or blood urea nitrogen occurred. Two patients experienced a decrease in hematocrit, which was not explained by blood loss or increase in plasma volume. Mean plasma volume was 3,713 +/- 129 ml (55.1 +/- 1.5 ml per kg ideal body weight) preparacentesis and 3,684 +/- 136 ml postparacentesis, the difference being -0.78% (p = 0.48, NS). Our results suggest that 5-liter paracentesis in nonedematous patients with tense portal hypertension-related ascites improves patient comfort and is not associated with a decrease in measured plasma volume.
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PMID:Large-volume paracentesis in nonedematous patients with tense ascites: its effect on intravascular volume. 335

The present article examines the relations among self-reported and physician-estimated chest pain variables to angiographically determined coronary stenosis (CAD) and neuroticism scores. Six of the 48 chest pain variables were significantly related to coronary stenosis, but only one variable, chest pain elicited by walking, was positively related to stenosis. Chest pain during sleep, sighing and dizziness accompanying chest pain, right lower chest pain radiation, and infrequent rest to cope with the chest pain were significantly negatively related to stenosis. Neuroticism scores (N) were not significantly related to CAD but were significantly correlated with 13 of the 48 chest pain variables. In addition to correlating positively with the chest pain variables that were negatively correlated with CAD, N scores were significantly related to higher pain severity ratings, being angry, annoyed, tense, afraid, worried, and upset before the chest pain, breathlessness during the pain episode, and pain sensations described as stabbing. The six chest pain variables significantly correlated with CAD yielded a multiple correlation of 0.58, accounting for 34% of the variance, whereas N scores accounted for only 5% of the variance; however, N contributed less than 1% unique variation to stenosis in combination with the six chest-pain variables. That N influences chest pain reports more than actual stenosis is further confirmed by the results of physicians' ratings of their patients' typical chest pain episodes. Recognition of patients' characteristic levels of distress or neuroticism may aid physicians in evaluating symptoms more accurately and in treating their chest pains more appropriately.
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PMID:The relation of chest pain symptoms to angiographic findings of coronary artery stenosis and neuroticism. 400 Dec 86

Under the name of RIED syndrome (retrocochlear inhibition efferent deafness syndrome). I would like to introduce the clinical subject of sudden neurosensorial deafness, or rapidly progressive deafness, which is accompanied by tinnitus, and occasionally by dizziness; all this is related to stressful situations, undergone by tense and perfectionist people who are unable to relax. The possible mechanism of this active efferent inhibition can work whether in one or both ears, from the cortex, going through the medial geniculate body to the low colliculus, and from here to the olivary cochlear set, whose efferent neurons terminate in the external ciliate cells. The possible way in which this efferent retrocochlear inhibition might work, could be in altering the usual operation of the external ciliate cells, which would stop their amplifying and modulating function, causing deafness and tinnitus; this possibly happens because of the immediate alteration of the signal that originates in the internal ciliate cells and that reaches the cortex through the afferent canal; due to the personality of the people that suffer from retrocochlear inhibition efferent deafness syndrome, it is assumed that there could be some disorder in the neurotransmitters; this is very similar to what happens with endogenous depression, and this malfunctioning would take place in the medial geniculate nuclei and inferior colliculus, disturbing the efferent control that this has over the cochlea.
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PMID:[Deafness syndrome caused by efferent retrocochlear inhibition]. 810 33

In an open study, we determined whether there were sex differences in the mood ratings of non-deprived light smokers and nonsmokers under baseline conditions and after completing a battery of cognitive tests that were mildly stressful. Male and female students who were light smokers (5-12 cigarettes a day) were tested immediately after smoking their usual cigarette, at a time that they normally smoked. They were compared with a group of male and female students who were nonsmokers and did not differ on age, IQ, personality measures, anxiety or depression. Compared with the nonsmokers, both male and female smokers felt overall significantly more discontented, troubled, tense, quarrelsome, furious, impatient, hostile, annoyed and disgusted and experienced greater dizziness. The performance of distracting cognitive tasks did not reveal anxiolytic effects of smoking, and after performance of these tasks, both smokers and nonsmokers became more discontented and anxious. In addition, after the cognitive testing, both male and female smokers showed greater increases than nonsmokers in feeling spiteful, rebellious, incompetent and in sweating, suggesting that they experienced greater mood changes in response to cognitive stress. There were no overall differences between the smokers and nonsmokers in the performance of divided or sustained attention tasks or in episodic memory. It is unlikely that either nicotine withdrawal or differences in cognitive performance could account for the greater anxiety, discontent and aggressive mood that was found in smokers.
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PMID:Mood differences between male and female light smokers and nonsmokers. 1217 65