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Target Concepts:
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Query: UMLS:C0002871 (
anemia
)
52,094
document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)
Thirty-two cats referred to the Feline Studies Centre between June 1987 and October 1988, and 14 in-contact cats, were found to be infected with feline immunodeficiency virus. Most of the 46 cats were non-pedigree and free ranging; 27 were male (19 neutered) and 19 were female (18 neutered). Their ages ranged from one to 17 years and the average age was 5.8 years. The most common clinical signs were lethargy, inappetence, weight loss, pyrexia and lymphadenopathy; most cases had multiple abnormalities. Other common signs were gingivitis, diarrhoea, rhinitis and ocular discharge. Eight cats had neoplasia. The commonest haematological abnormalities were
anaemia
, neutropenia, lymphopenia and monocytosis. Eight cats had lymphocytosis; seven of these were in a single house-hold. Several cats had high serum globulin levels and half of those tested had high IgG levels. Seven cats had no detectable antibody to feline immunodeficiency virus even though the virus was cultured from the peripheral blood lymphocytes. During follow-up for up to 60 weeks one cat died and 23 were destroyed on humane grounds.
Vet
Rec
1989 Sep 23
PMID:Clinical and laboratory findings in cats infected with feline immunodeficiency virus. 255 57
Three domestic short-haired cats with a history of anorexia and loss of body condition had high rectal temperatures, and a normocytic, normochromic
anaemia
. Two of them were also dyspnoeic, and thoracic radiographs revealed a diffuse, unstructured increase in radio-opacity involving all the lung lobes. Examination of Giemsa-stained blood smears and culture of blood monocytes revealed purplish-staining intracytoplasmic inclusions, in monocytes and lymphocytes, which occurred either singly or in aggregates. Electron micrographs of a buffy coat smear from one of the cats revealed round intracytoplasmic inclusions, with electron dense and lucid areas morphometrically similar to those found in other members of the genus Ehrlichia. An attempt to culture chlamydia from one of the cats was unsuccessful. The cats were treated successfully, one with tetracycline hydrochloride and the other two with imidocarb dipropionate.
Vet
Rec
1989 Oct 21
PMID:Feline anaemia associated with Ehrlichia-like bodies in three domestic short-haired cats. 258 53
Haemobartonella felis infection was demonstrated in 38 cats which could be divided into four groups as follows: group A, feline leukaemia virus (FeLV) free cats with H felis infection alone; group B, FeLV free cats with H felis infection and other clinical conditions; group C, FeLV positive cats with H felis infection but no clinical manifestation of FeLV related or any other intercurrent disease; and group D, FeLV positive cats with H felis infection and clinical manifestations of FeLV related or other diseases. Cats in group A were healthy carriers of the infection and none was anaemic, whereas some in group B had clinical haemobartonellosis and
anaemia
. This
anaemia
was mainly mild, normocytic and normochromic. Most of the cats in group C and all in group D were more severely ill and anaemic, the
anaemia
usually being macrocytic and hypochromic. Splenomegaly occurred only in groups C and D. Treatment with tetracyclines did not eliminate H felis from any of the cats and blood transfusions were ineffective in promoting long term recovery from
anaemia
in cats with intercurrent H felis and FeLV infections. The findings in the cats in groups C and D were further compared with those in a fifth group of cats which were infected with FeLV but free of H felis.
Vet
Rec
1988 Jan 09
PMID:Feline haemobartonellosis: clinical, haematological and pathological studies in natural infections and the relationship to infection with feline leukaemia virus. 283 61
An outbreak of equine viral arteritis in the American state of Kentucky led to a temporary ban being imposed by France, Ireland and the UK on the importation of horses from the USA during 1984. Sporadic cases of influenza caused by the type 2 strain of the virus were confirmed in France throughout the year and cases of strangles in thoroughbred mares and foals were reported from all three countries. No cases of contagious equine metritis were confirmed among the thoroughbred breeding population, although a number of outbreaks of the abortion form of rhinopneumonitis occurred. A small number of clinical cases of equine infectious
anaemia
were confirmed in France.
Vet
Rec
1985 Feb 09
PMID:Infectious disease incidence among horses in France, Ireland and the United Kingdom during 1984. 298 33
Six 34- to 42-day-old lambs raised in coccidia-free conditions were inoculated with 70,000 sporocysts derived from sheep heart with microscopic sarcocysts. Fever and mild anorexia occurred between 25 and 33 days after inoculation. A transient
anaemia
was most marked 32 days after inoculation. Lambs were killed and examined 14, 25, 33, 42, 60 and 81 days after inoculation. Gross lesions were absent. First and second generation meronts were present in endothelial cells at 25 and 33 days after inoculation. Meronts were most numerous in kidney glomeruli. Developing sarcocysts were rare at 42 days after inoculation. Sarcocysts with a primary cyst wall 2 to 3 micron thick composed of palisade projections were common at 60 and 81 days after inoculation in striated muscle and brain. Mild to severe striated muscle myositis and non-suppurative encephalitis or encephalomyelitis with glial nodules were observed 25 to 81 days after inoculation. Sarcocyst frequency varied considerably; it was highest in myocardium, M vastus intermedius, M vastus medialis, M extensor carpi radialis and tongue muscle and was lowest in M masseter.
Vet
Rec
1986 Nov 22
PMID:Experimental microcyst sarcocystis infection in lambs: pathology. 310 Dec 71
Blood counts of healthy juvenile and adult llamas (Lama glama) and guanacoes (L guanacoe) showed that guanacoes have higher red cell counts, haemoglobin values and packed cell volumes than llamas. In both species, the numbers of lymphocytes and platelets were higher in juveniles than in adults. By reference to the values found in normal animals, abnormal haematological variations were detected in a number of sick individuals. Neutrophilia, hyperfibrinogenaemia and a tendency to develop regenerative hypochromic
anaemia
were observed in animals with acute and chronic inflammatory diseases and hypochromic macrocytic anaemia occurred in animals with parasitic infections. Many individuals in which subclinical intestinal parasitic infections were suspected had relatively high eosinophil counts although their other haematological values were normal.
Vet
Rec
1988 Mar 05
PMID:Haematology of clinically normal and abnormal captive llamas and guanacoes. 336 14
The myeloproliferative diseases may present with a variety of clinical signs including regenerative or non-regenerative anaemias, bleeding diatheses, septicaemia or fever of unknown origin. These signs will raise suspicions of myeloproliferative disease but such disease may also be an incidental finding on routine haematological examination. In either case a bone marrow biopsy will be required for confirmation. Investigation for other causes of
anaemia
, haemostatic dysfunction or other causes of white cell abnormalities is important in animals where the peripheral blood and bone marrow findings are equivocal or atypical of myeloproliferative disease. Treatment of acute myeloproliferative diseases is presently impractical in veterinary medicine. Therapy of the chronic myeloproliferative diseases depends upon the suppression of the proliferation of the affected clones together with attention to the secondary effects of the disease and to the adverse effects of therapy.
Vet
Rec
1987 Nov 21
PMID:Myeloproliferative disease in the dog and cat: clinical presentations, diagnosis and treatment. 342 33
Infectious stunting is recorded in detail in three egg-type pullet flocks and one egg-type breeder flock. Three different strains of brown egg birds were involved. The clinical effects were less severe than those usually seen in broilers. Excessive mortality during the first three weeks was a feature of all four flocks. Fibrous atrophy of the pancreas was seen in one flock and could still be found in individual birds at 18 weeks old. Bone disorders were seen in two flocks. Severe
anaemia
was seen in some birds in one flock. Severe emaciation of the pectoral muscles was an unexplained and prominent feature of all the flocks. As the birds grew the visible and clinical evidence of stunting was much reduced and it appeared that compensatory growth took place so that by 18 weeks old the flocks appeared normal and the culling rate at caging was negligible.
Vet
Rec
1986 Oct 04
PMID:Probable infectious stunting syndrome in replacement pullets. 378 93
The equine bone marrow responds to blood loss by increased erythropoiesis, only releasing reticulocytes into the peripheral circulation in severe chronic
anemia
. We have used morphometric analysis based on electron microscopy of the equine marrow to examine the maturation and release of reticulocytes. Developing red cells in the bone marrows of normal and chronically anemic horses were divided into four stages: early, intermediate, late-stage erythroblasts, and reticulocytes. Morphometric analysis of each stage included volume density of mitochondria per micron3 of cytoplasm, surface area of the outer mitochondrial membrane per unit volume of mitochondria, and the number of ribosomes per unit volume of cytoplasm (total, clustered, single). Matched t tests between normal and anemic animals showed significant differences (P less than or equal to .001) for volume density of mitochondria and numbers of ribosomes only at the reticulocyte stage. The large reticulocyte produced and released in chronic
anemia
may be best explained by a skipped mitotic division.
Anat
Rec
1985 Oct
PMID:Equine bone marrow: a quantitative analysis of erythroid maturation. 407 71
Zinc toxicity, copper deficiency and an associated
anaemia
among weaned swill-fed pigs is described. The source of zinc was flaking galvanising from the inside of bins used to store swill before processing. The disease condition readily responded to supplementation of the diet with copper sulphate. The interactions between dietary zinc, copper and calcium are discussed with reference to the swill-feeding industry. It is concluded that supplementary copper should be routinely included in the ration of swill-fed pigs and that the use of galvanised bins for storing swill should be avoided.
Vet
Rec
1985 Nov 23
PMID:Zinc toxicity, copper deficiency and anaemia in swill-fed pigs. 408 24
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