Gene/Protein Disease Symptom Drug Enzyme Compound
Pivot Concepts:   Target Concepts:
Query: EC:3.6.1.3 (ATPase)
 65,361 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

Some molecular and cellular mechanisms of compensation for respiratory failure (RF) were examined in 90 patients with nonspecific pulmonary diseases. In acute RF, the content of 2,3-DPH in red blood cells was increased. As RF progressed, the compensatory-adaptation reaction became less marked. In chronic RF, the 2,3-DPH content in red blood cells did not differ from the control values. The disease was characterized by cholesterol a and sphingomyelin accumulation, a reduction in red blood cell ATPase activity, and an increase in microviscosity of red blood cell membranes. Decelerated utilization of ATPases in red blood cells is of a role in the genesis of a rise of the 2,3-DPH content in acute RF.
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PMID:[Molecular and cellular mechanisms of compensation for respiratory failure in nonspecific lung diseases]. 315 20