Gene/Protein
Disease
Symptom
Drug
Enzyme
Compound
Pivot Concepts:
Gene/Protein
Disease
Symptom
Drug
Enzyme
Compound
Target Concepts:
Gene/Protein
Disease
Symptom
Drug
Enzyme
Compound
Query: EC:2.3.3.1 (
citrate synthase
)
4,488
document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)
The present experiment was designed to assess the effects of seed soaking with 24-epibrassinolide (EBR) on the physiology of
Brassica juncea
L. seedlings grown under imidacloprid (IMI) toxicity. Application of EBR increased the length of seedlings, dry weight, and pigment contents, polyphenols, total phenols, and organic acids under IMI toxicity. The expression of genes coding key enzymes of pigment, phenols, polyphenols, and organic acid biosynthetic pathways was also studied including
CHLASE
(chlorophyllase),
PSY
(
phytoene synthase
),
CHS
(chalcone synthase) and
PAL
(phenylalanine ammonialyase),
CS
(
citrate synthase
),
SUCLG1
(succinyl Co-A ligase,),
SDH
(succinate dehydrogenase),
FH
(fumarate hydratase),
MS
(malate synthase). Multiple linear regression (MLR) analysis revealed that IMI application regressed negatively on seedling length, dry weight and total chlorophyll content. However, EBR seed treatment regressed positively on all the parameters studied. Moreover, interaction between IMI and EBR showed positive regression for growth parameters, content of pigments, total polyphenol, total phenol and malate, and expression of
PSY
and
PAL
. Negative interactions were noticed for the contents of fumarate, succinate and citrate, and expression of
CHS
and all genes studied related to organic acid metabolism. In conclusion, EBR enhanced the growth and contents of all studied metabolites by regulating the gene expression of
B. juncea
seedlings under IMI stress.
...
PMID:Pre-sowing Seed Treatment with 24-Epibrassinolide Ameliorates Pesticide Stress in
Brassica juncea
L. through the Modulation of Stress Markers. 2785 60
