Gene/Protein Disease Symptom Drug Enzyme Compound
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Query: EC:1.6.5.4 (SOR)
 720 document(s) hit in 31,850,051 MEDLINE articles (0.00 seconds)

ABA regulation of antioxidant activity during post-germination desiccation and subsequent rehydration was studied in two wheat cultivars PBW 644 (ABA-higher sensitive and drought tolerant) and PBW 343 (ABA-lesser sensitive and drought susceptible) where 1 d-germinated seeds were exposed to ABA/ PEG- 6000 for next 1 d, desiccated for 4 d and subsequently rehydrated for 4 d. Ascorbate, dehydrascorbate to ascorbate ratio, malondialdehyde (MDA), hydroxyl radicals, and activities of monodehydroascorbate reductase (MDHAR), dehydroascorbate reductase (DHAR), alcohol dehydrogenase (AlcDH) and aldehyde dehydrogenase (AldDH) were measured in seedlings just before desiccation (2 d old), desiccated (6 d old) and rehydrated (10 d old) stages. ROS/NO signaling was studied under CT and ABA supply by supplying ROS and NO scavengers. During desiccation, both cultivars showed increase of oxidative stress (dehydroascorbate to ascorbate ratio, MDA, hydroxyl radicals) and antioxidant activity in the form of ascorbate content and AldDH activity while other antioxidant enzymes were not increased. PBW 644 showed higher antioxidant activity thus produced less oxidative stress compared to PBW 343. During rehydration, activities of all antioxidant enzymes and levels of ROS (hydroxyl radicals) were increased in both cultivars and MDA was decreased in PBW 343. ABA supply improved desiccation as well as rehydration by improving all parameters of antioxidant activity tested in this study. PEG supply resembled to ABA-supply for its effects. ABA/PEG improvements were seen higher in PBW 644. ROS/NO-signalling was involved under CT as well as under ABA for increasing antioxidant activity during desiccation as well as rehydration in both cultivars.
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PMID:ABA regulation of antioxidant activity during post-germination desiccation and subsequent rehydration in wheat. 3025 77

This study investigated the changes in enzyme activity and gene expression in reactive oxygen species (ROS) and fatty acid metabolism in Docteur Jules Guyot pears after acibenzolar-S-methyl (ASM) treatment to elucidate the role of ROS and fatty acid metabolism in senescence. The results demonstrated that applying ASM postharvest significantly suppressed H2O2 content and enhanced catalase and superoxide dismutase activities in pears. Ascorbate peroxidase, glutathione reductase, dehydroascorbate reductase, and monodehydroascorbate reductase activities and the reduced glutathione content in pears were also induced by ASM. Postharvest ASM dipping remarkably enhanced PcSOD, PcCAT, PcAPX, and PcDHAR expressions and fatty acid synthetase activity in pears. Postharvest applying ASM significantly decreased malondialdehyde content and lipoxygenase, hydroperoxidelyase, alcohol dehydrogenase, and alcohol acyltransferase activities in pears. ASM distinctly inhibited PcPLD, PcLOX, PcHPL, PcADH, and PcAAT expressions in pears. The findings suggest that postharvest applying ASM could modulate ROS and fatty acid metabolism to delay senescence in pears.
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PMID:Postharvest Application of Acibenzolar-S-methyl Delays the Senescence of Pear Fruit by Regulating Reactive Oxygen Species and Fatty Acid Metabolism. 3227 67